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Harry A. Quigley
Researcher at Johns Hopkins University
Publications - 506
Citations - 72465
Harry A. Quigley is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Glaucoma & Intraocular pressure. The author has an hindex of 115, co-authored 492 publications receiving 65186 citations. Previous affiliations of Harry A. Quigley include University of Miami & Johns Hopkins University School of Medicine.
Papers
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Journal ArticleDOI
Cataract after glaucoma filtration surgery.
Camille Hylton,Nathan Congdon,David S. Friedman,John H. Kempen,Harry A. Quigley,Eric B Bass,Henry D. Jampel +6 more
TL;DR: Patients undergoing glaucoma surgery are at increased risk for the development and progression of cataract, according to a systematic review of relevant studies published in English.
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Incidence of acute angle-closure glaucoma after pharmacologic mydriasis.
Ketan H. Patel,Jonathan C. Javitt,Jonathan C. Javitt,James M. Tielsch,Debra A. Street,Joanne Katz,Harry A. Quigley,Alfred Sommer +7 more
TL;DR: When the three screening factors were considered alone and in combination, the most effective combination for predicting a potentially occludable angle was to use shallow anterior chamber on penlight examination and history of glaucoma.
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Study of central retinal ganglion cell loss in experimental glaucoma in monkey eyes.
TL;DR: Foveal ganglion cells appear to be susceptible to experimental glaucoma injury and psychophysical testing of foveal functions may deserve renewed attention in glAUcoma diagnosis and follow-up.
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Clinical trials for glaucoma neuroprotection are not impossible.
TL;DR: With design features discussed in the review, neuroprotection trials can be effectively carried out and new glaucoma treatments known as neuroprotection can be developed.
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Topical timolol decreases plasma high-density lipoprotein cholesterol level.
Anne L. Coleman,Anne L. Coleman,D. L. Chris Diehl,Henry D. Jampel,Paul S. Bachorik,Harry A. Quigley +5 more
TL;DR: Higher baseline high- density lipoprotein cholesterol levels were associated with larger reductions in high-density lipop Protein cholesterol levels, and higher baseline cholesterol-high-density cholesterol ratio increased 8%, suggesting that baseline cholesterol levels may have been affected by beta-Adrenergic antagonists taken orally.