H
Heather Dickinson-Anson
Researcher at University of California, Irvine
Publications - 15
Citations - 4307
Heather Dickinson-Anson is an academic researcher from University of California, Irvine. The author has contributed to research in topics: Basal forebrain & Midazolam. The author has an hindex of 14, co-authored 15 publications receiving 4131 citations. Previous affiliations of Heather Dickinson-Anson include Salk Institute for Biological Studies.
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Neurogenesis in the dentate gyrus of the adult rat: age-related decrease of neuronal progenitor proliferation
TL;DR: It is confirmed that in the adult rat brain, neuronal progenitor cells divide at the border between the hilus and the granule cell layer (GCL) and in adult rats, the progeny of these cells migrate into the GCL and express the neuronal markers NeuN and calbindin-D28k.
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Altered synaptic physiology and reduced susceptibility to kainate-induced seizures in GluR6-deficient mice
Christophe Mulle,Andreas W. Sailer,Isabel Pérez-Otaño,Heather Dickinson-Anson,Pablo E. Castillo,Ingrid Bureau,Cornelia Maron,Fred H. Gage,Jeffrey R. Mann,Bernhard Bettler,Stephen F. Heinemann +10 more
TL;DR: It is found that GluR6-deficient mice are less susceptible to systemic administration of kainate, as judged by onset of seizures and by the activation of immediate early genes in the hippocampus.
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Behavioral and neuroanatomical investigation of Highly Superior Autobiographical Memory (HSAM).
Aurora K. R. LePort,Aaron T. Mattfeld,Heather Dickinson-Anson,James H. Fallon,Craig E.L. Stark,Frithjof Kruggel,Larry Cahill,James L. McGaugh +7 more
TL;DR: Findings indicated that HSAM participants performed significantly better at recalling public as well as personal autobiographical events aswell as the days and dates on which these events occurred, but their performance was comparable to age- and sex-matched controls on most standard laboratory memory tests.
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Central neuronal loss and behavioral impairment in mice lacking neurotrophin receptor p75.
TL;DR: Vulnerability to the lack of p75 is demonstrated in adult central neurons that are neurotrophin dependent and the loss of noncholinergic central neurons in mice lacking p75 suggests a role for p75 in cell survival by an as yet undetermined mechanism.
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Generation and analysis of GluR5(Q636R) kainate receptor mutant mice.
Andreas W. Sailer,Geoffrey T. Swanson,Isabel Pérez-Otaño,Lora O’Leary,Shelle Malkmus,Richard H. Dyck,Heather Dickinson-Anson,Hans H. Schiffer,Cornelia Maron,Tony L. Yaksh,Fred H. Gage,Stephen O'Gorman,Stephen F. Heinemann +12 more
TL;DR: Investigations with the GluR5-editing mutant mice have defined a set of physiological processes in which editing of the GLUR5 subunit is unlikely to play an important role, and the role of glutamate receptor mRNA editing in the brain is addressed.