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Hojjatallah Alaei

Researcher at Isfahan University of Medical Sciences

Publications -  121
Citations -  1921

Hojjatallah Alaei is an academic researcher from Isfahan University of Medical Sciences. The author has contributed to research in topics: Morphine & Internal medicine. The author has an hindex of 23, co-authored 104 publications receiving 1569 citations.

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Probiotics treatment improves diabetes-induced impairment of synaptic activity and cognitive function: behavioral and electrophysiological proofs for microbiome-gut-brain axis

TL;DR: It is concluded that probiotics efficiently reverse deteriorated brain functions in the levels of cognitive performances and their proposed synaptic mechanisms in diabetes mellitus.
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Treadmill running reverses retention deficit induced by morphine.

TL;DR: It is concluded that exercise increased the delay time of entry to the dark electrical foot shock box, suggesting that morphine impaired the short-term memory and learning and this was reversed by the treadmill running.
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Treadmill exercise reduces self-administration of morphine in male rats.

TL;DR: As exercise can activate many neurotransmitter systems involved in the addiction process and increase the release of endorphins, it is likely that the effect of short-term and mid-term exercises on the self-administration of morphine in rats will decrease.
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Effects of treadmill running on spatial learning and memory in streptozotocin-induced diabetic rats.

TL;DR: The present results suggest that spatial learning and memory is affected under diabetic conditions and that treadmill running prevents these effects, and correspond to the possibility that treadmillRunning is helpful in the prevention and alleviation of the cognitive decline in diabetes mellitus.
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Treadmill running improves spatial memory in an animal model of Alzheimer's disease.

TL;DR: The present results suggest that spatial memory is affected under Alzheimer conditions and that treadmill running improves these effects and is suggested to contribute to the alleviation of the cognitive decline in AD.