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Holly A. Shill

Researcher at National Institutes of Health

Publications -  8
Citations -  499

Holly A. Shill is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Dystonia & Anti-gliadin antibodies. The author has an hindex of 6, co-authored 8 publications receiving 483 citations.

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Regional cerebral blood flow correlates of the severity of writer's cramp symptoms.

TL;DR: Overactivity of SI is more dramatic and suggests a primary deficit in processing sensory feedback in writer's cramp, which may arise in part as a dysfunction of sensory circuits, which causes defective sensorimotor integration resulting in co-contractions of muscles and overflow phenomena.
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Immune Cross-Reactivity in Celiac Disease: Anti-Gliadin Antibodies Bind to Neuronal Synapsin I

TL;DR: This study characterized the binding of affinity-purified anti-gliadin Abs from immunized animals to brain proteins by one- and two-dimensional gel electrophoresis, immunoblotting, and peptide mass mapping, and identified the major immunoreactive protein as synapsin I.
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Motor training as treatment in focal hand dystonia.

TL;DR: A motor training program for individualized finger movements aimed at decreasing abnormal overflow of movement to fingers not involved in a task led to mild subjective improvement and some improvement in handwriting, but it was not sufficient to reverse motor cortex abnormalities measured by TMS and EEG.
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Generators of movement-related cortical potentials: fMRI-constrained EEG dipole source analysis.

TL;DR: To clarify the precise location and timing of the cortical activation in voluntary movement, dipole source analysis integrating multiple constraints wa conducted for the movement-related cortical potentia (MRCP) and deduced patterns of activation similar to that of the bilateral precentral gyri.
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Respiratory complications of Parkinson's disease.

TL;DR: Overtreatment with levodopa causes respiratory dyskinesia that may be difficult to differentiate from complications of the disease itself, and therapy for Parkinson's may also contribute to pulmonary morbidity.