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Khalaf Bushara

Researcher at University of Minnesota

Publications -  62
Citations -  2669

Khalaf Bushara is an academic researcher from University of Minnesota. The author has contributed to research in topics: Ataxia & Spinocerebellar ataxia. The author has an hindex of 23, co-authored 50 publications receiving 2250 citations. Previous affiliations of Khalaf Bushara include University of Wisconsin Hospital and Clinics & Veterans Health Administration.

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Multimodal imaging of brain reorganization in motor areas of the contralesional hemisphere of well recovered patients after capsular stroke

TL;DR: Effective recovery is based on enhanced utilization of ipsi- and contralesional resources, basic corticospinal commands arise from the lesioned hemisphere without recruitment of ('latent') uncrossed corticOSPinal tract fibres and increased contralsional activity probably facilitates control of recovered motor function by operating at a higher-order processing level, similar to but not identical with the extended network concerned with complex movements in healthy subjects.
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Enhanced Excitability of the Human Visual Cortex Induced by Short-term Light Deprivation

TL;DR: A substantial increase in visual cortex excitability was demonstrated after a short period of light deprivation, which may underlie behavioral gains reported in humans and animals associated with light deprivation.
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Botulinum toxin--a possible new treatment for axillary hyperhidrosis.

TL;DR: To determine the dosage, pattern and duration of the anhidrotic effect of botulinum toxin and to test the efficacy of axillary injections, seven healthy volunteers were studied.
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Sialorrhea in amyotrophic lateral sclerosis: a hypothesis of a new treatment - botulinum toxin A injections of the parotid glands

TL;DR: Injection of the parotid glands with botulinum toxin is proposed as an new treatment for sialorrhea in patients with amyotrophic lateral sclerosis and other neurological diseases.
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Regional cerebral blood flow correlates of the severity of writer's cramp symptoms.

TL;DR: Overactivity of SI is more dramatic and suggests a primary deficit in processing sensory feedback in writer's cramp, which may arise in part as a dysfunction of sensory circuits, which causes defective sensorimotor integration resulting in co-contractions of muscles and overflow phenomena.