H
Hongxian Yu
Researcher at Northeast Forestry University
Publications - 8
Citations - 360
Hongxian Yu is an academic researcher from Northeast Forestry University. The author has contributed to research in topics: Oxidative stress & Apoptosis. The author has an hindex of 7, co-authored 8 publications receiving 177 citations.
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Journal ArticleDOI
ROS-Induced Hepatotoxicity under Cypermethrin: Involvement of the Crosstalk between Nrf2/Keap1 and NF-κB/iκB-α Pathways Regulated by Proteasome.
TL;DR: In this article, the toxic phenotype and detoxification mechanism in grass carp (Ctenopharyngodon idella) after treatment with waterborne permethrin (0.651 μg/L) for 6 weeks in vivo or 6.392 μM for 24 hours in vitro.
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Environmentally relevant concentration of cypermethrin or/and sulfamethoxazole induce neurotoxicity of grass carp: Involvement of blood-brain barrier, oxidative stress and apoptosis.
TL;DR: The potential co-ecological risks of pesticide and antibiotic in the aquatic organism will be revealed, and basic data for their safety and risk assessment will be provided.
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The cardiotoxicity of the common carp (Cyprinus carpio) exposed to environmentally relevant concentrations of arsenic and subsequently relieved by zinc supplementation.
TL;DR: The interaction of divalent zinc ion (Zn2+), an efficient reactive oxygen species (ROS) scavenger with arsenite in the heart of common carp is explored, and the application of zinc preparations may provide a candidate for the prevention and treatment for arsenic poisoning.
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Grass carps co-exposed to environmentally relevant concentrations of cypermethrin and sulfamethoxazole bear immunodeficiency and are vulnerable to subsequent Aeromonas hydrophila infection.
TL;DR: By affecting the conduction of TLR signaling pathway, CMN or/and SMZ exposure inhibits the innate immune response of fish and reducing their disease resistance, highlighting the importance of rational and regulated use of these pesticides and antibiotics.
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Destruction of redox and mitochondrial dynamics co-contributes to programmed cell death in chicken kidney under arsenite or/and copper (II) exposure.
TL;DR: The results highlighted the need to take precautions against copper and arsenic co-exposure when considering their impact in susceptible animals/populations and can function independently or cooperatively to affect oxidative stress, mitochondrial dynamics and programmed cell death.