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Horst Buerger

Researcher at University of Münster

Publications -  118
Citations -  9624

Horst Buerger is an academic researcher from University of Münster. The author has contributed to research in topics: Breast cancer & Cancer. The author has an hindex of 51, co-authored 118 publications receiving 9054 citations. Previous affiliations of Horst Buerger include University of Hamburg & Utrecht University.

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MALAT-1, a novel noncoding RNA, and thymosin beta4 predict metastasis and survival in early-stage non-small cell lung cancer.

TL;DR: The identification of MALAT-1 emphasizes the potential role of noncoding RNAs in human cancer and contributes to the identification of early-stage NSCLC patients that are at high risk to develop metastasis.
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Silver-coated megaendoprostheses in a rabbit model--an analysis of the infection rate and toxicological side effects.

TL;DR: The new silver-coated Mutars-megaprosthesis resulted in reduced infection rates without toxicological side effects, suggesting that this prosthesis might be a promising device in tumor surgery exhibiting antimicrobial activity.
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Comparative genomic hybridization of ductal carcinoma in situ of the breast-evidence of multiple genetic pathways.

TL;DR: Analysis of paraffin‐embedded specimens of DCIS and six associated invasive carcinomas revealed a genetic pattern almost identical to the one seen in the DCIS counterpart, characterize DCIS as a genetically far‐advanced, heterogeneous lesion and as a direct precursor of invasive breast cancer.
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Lack of toxicological side-effects in silver-coated megaprostheses in humans.

TL;DR: The silver-coated megaprosthesis allowed a release of silver without showing any local or systemic side-effects and can be considered as non-toxic.
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Different genetic pathways in the evolution of invasive breast cancer are associated with distinct morphological subtypes.

TL;DR: Data demonstrate the close genetic similarity of well‐, intermediately, and poorly differentiated DCIS and distinct morphological types of invasive breast carcinoma, providing further evidence that DCIS is a direct precursor lesion ofvasive breast cancer and that various evolutionary genetic pathways exist.