H
Howard N. Hodis
Researcher at University of Southern California
Publications - 318
Citations - 24700
Howard N. Hodis is an academic researcher from University of Southern California. The author has contributed to research in topics: Menopause & Intima-media thickness. The author has an hindex of 74, co-authored 297 publications receiving 22233 citations. Previous affiliations of Howard N. Hodis include Oklahoma Medical Research Foundation.
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Journal ArticleDOI
Relationship between Serum Levels of Sex Hormones and Progression of Subclinical Atherosclerosis in Postmenopausal Women
TL;DR: Estrogen and SHBG are associated with reduced subclinical atherosclerosis progression in healthy postmenopausal women and are partially mediated by their beneficial effects on lipids.
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Cytomegalovirus Immunoglobulin G Antibody Is Associated With Subclinical Carotid Artery Disease Among HIV-Infected Women
Christina M. Parrinello,Elizabeth Sinclair,Alan L. Landay,Nell S. Lurain,A. Richey Sharrett,Stephen J. Gange,Xiaonan Xue,Peter W. Hunt,Steven G. Deeks,Howard N. Hodis,Robert C. Kaplan +10 more
TL;DR: Cytomegalovirus antibody titers are increased in HIV-infected women and associated with subclinical cardiovascular disease and host responses to CMV may be abnormal in HIV infection andassociated with clinical disease.
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Correlations between measures of atherosclerosis change using carotid ultrasonography and coronary angiography
Wendy J. Mack,Laurie LaBree,Chao-ran Liu,Chi-Hua Liu,Robert H. Selzer,Robert H. Selzer,Howard N. Hodis +6 more
TL;DR: While measures obtained by carotid ultrasonography and coronary angiography are correlated, they each assess different aspects of atherosclerosis change.
Journal Article
The lipid, lipoprotein, and apolipoprotein effects of hydroxychloroquine in patients with systemic lupus erythematosus.
TL;DR: Results are encouraging in that hydroxychloroquine, in addition to being useful for alleviating the primary symptoms of SLE, may also be useful for ameliorating the adverse effects of corticosteroid therapy on triglyceride-rich lipoprotein metabolism.
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Analysis of plasma cholesterol oxidation products using gas- and high-performance liquid chromatography/mass spectrometry
TL;DR: Although several of the plasma cholesterol oxides may derive from cholesterol-7-hydroperoxides, it appears that the latter are either unstable and decompose in plasma, are metabolized to other cholesterol oxidation products, or break down during their isolation.