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Hua Feng

Researcher at Third Military Medical University

Publications -  339
Citations -  13117

Hua Feng is an academic researcher from Third Military Medical University. The author has contributed to research in topics: Medicine & Intracerebral hemorrhage. The author has an hindex of 38, co-authored 298 publications receiving 10585 citations. Previous affiliations of Hua Feng include Chongqing University & Southwest General Health Center.

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Glioma-initiating cells: a predominant role in microglia/macrophages tropism to glioma.

TL;DR: A predominant role of GICs in microglia/macrophages tropism to glioma and a close positive correlation between the distribution of G ICs and TAM/Ms are reported, indicating an important part of cancer-related inflammation, which may participate in the architecture of the GIC niche.
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Terahertz pulsed spectroscopy of paraffin-embedded brain glioma

TL;DR: The results indicate that paraffin-embedded brain gliomas have a higher refractive index, absorption coefficient, and dielectric constant than normal brain tissue.
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Glial scar and neuroregeneration: histological, functional, and magnetic resonance imaging analysis in chronic spinal cord injury.

TL;DR: A critical time window of postinjury recovery and a detailed spatial orientation of glial scar are revealed, which would provide an important basis for the development of therapeutic strategy for glial Scar ablation.
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Norrin Protected Blood–Brain Barrier Via Frizzled-4/β-Catenin Pathway After Subarachnoid Hemorrhage in Rats

TL;DR: Norrin protected blood–brain barrier integrity and improved neurological outcome after SAH, and the action of Norrin appeared mediated by Frizzled-4 receptor activation, which promoted &bgr;-catenin nuclear translocation, which then enhanced Occludin, VE-Cadherin, and ZO-1 expression.
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Endogenous hydrogen sulphide attenuates NLRP3 inflammasome-mediated neuroinflammation by suppressing the P2X7 receptor after intracerebral haemorrhage in rats.

TL;DR: Results indicated endogenous H2S synthesis was impaired after ICH, which plays a pivotal role in the P2X7R/NLRP3 inflammasome-associated neuroinflammatory response in the pathogenesis of secondary brain injury.