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Hua Feng

Researcher at Third Military Medical University

Publications -  339
Citations -  13117

Hua Feng is an academic researcher from Third Military Medical University. The author has contributed to research in topics: Medicine & Intracerebral hemorrhage. The author has an hindex of 38, co-authored 298 publications receiving 10585 citations. Previous affiliations of Hua Feng include Chongqing University & Southwest General Health Center.

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Interleukin-1 receptor associated kinases-1/4 inhibition protects against acute hypoxia/ischemia-induced neuronal injury in vivo and in vitro.

TL;DR: The results show that IRAK-1/4 inhibition plays a neuroprotective role in H/I-induced brain injury and attenuated CoCl(2)-induced cytotoxicity and apoptosis in B35 cells in vitro.
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High-sensitivity terahertz imaging of traumatic brain injury in a rat model.

TL;DR: It is demonstrated that different degrees of experimental traumatic brain injury (TBI) can be differentiated clearly in fresh slices of rat brain tissues using transmission-type terahertz (THz) imaging system, and THz imaging has great potential as an alternative method for the fast diagnosis of TBI.
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T lymphocytes infiltration promotes blood-brain barrier injury after experimental intracerebral hemorrhage.

TL;DR: In this article, the authors investigated the spatial-temporal distribution of infiltrating T lymphocytes after ICH in C57BL/6 mice by immunofluorescence and flow cytometry, and the accompanying change rules of BBB permeability were detected by Evans blue dye leakage and tight junction protein expression.
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Milk Fat Globule-Epidermal Growth Factor-8 Pretreatment Attenuates Apoptosis and Inflammation via the Integrin-β3 Pathway after Surgical Brain Injury in Rats.

TL;DR: It is found that rhMFGE8 pretreatment effectively alleviated neurological deficits and decreased brain water content and apoptotic cells in the SBI model through the MFGE8/integrin-β3 pathway, and treatment time was an important factor in achieving curative effects.
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Suppression of Akt1 phosphorylation by adenoviral transfer of the PTEN gene inhibits hypoxia-induced proliferation of rat pulmonary arterial smooth muscle cells

TL;DR: The findings suggest that phospho-PTEN loss in the nuclei of PASMCs under hypoxic conditions may be the major cause of aberrant activation of Akt1 and may, therefore, play an important role in hypoxia-associated pulmonary arterial remodeling.