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Hua Feng

Researcher at Third Military Medical University

Publications -  339
Citations -  13117

Hua Feng is an academic researcher from Third Military Medical University. The author has contributed to research in topics: Medicine & Intracerebral hemorrhage. The author has an hindex of 38, co-authored 298 publications receiving 10585 citations. Previous affiliations of Hua Feng include Chongqing University & Southwest General Health Center.

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Osteopontin Mediates Hyperbaric Oxygen Preconditioning-Induced Neuroprotection Against Ischemic Stroke.

Shengli Hu, +5 more
- 01 Aug 2015 - 
TL;DR: OPN plays an important role in the neuroprotection elicited by HBO-PC, and preconditioning with HBO promoted expression of OPN, which reduced the expression of interleukin (IL)-1β/nuclear factor-κ-gene binding and augmented protein kinase B (Akt).
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Cannabinoid CB2 receptor stimulation attenuates brain edema and neurological deficits in a germinal matrix hemorrhage rat model.

Yihao Tao, +8 more
- 30 Mar 2015 - 
TL;DR: A potential clinical utility for CB2R agonists as a potential therapy to reduce neurological injury and improve patient outcomes after GMH is suggested.
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Glycine Potentiates AMPA Receptor Function through Metabotropic Activation of GluN2A-Containing NMDA Receptors.

Lijun Li, +12 more
- 19 Oct 2016 - 
TL;DR: A metabotropic function of GluN2ARs is revealed in mediating glycine-induced potentiation of AMPA receptor function via ERK1/2 activation in cultured mouse hippocampal neurons.
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Intraparenchymal schwannoma of the medulla oblongata. Case report.

Jiangkai Lin, +4 more
- 01 Mar 2003 - 
TL;DR: A 48-year-old man presented with a rare intraparenchymal schwannoma of the medulla oblongata, which at the time was thought to be a glioma, became larger on magnetic resonance images and was resected through a suboccipital craniectomy via the transcerebellomedullary fissure approach.
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Scutellarin attenuates vasospasm through the Erk5-KLF2-eNOS pathway after subarachnoid hemorrhage in rats

Qiang Li, +9 more
- 01 Dec 2016 - 
TL;DR: Results indicate that SCU could attenuate vasospasm and neurological deficits via modulating the Erk5-KLF2-eNOS pathway after SAH, which may provide an experimental basis for the clinical use of SCU treatment in SAH patients.