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Hubert Mönnikes

Researcher at Charité

Publications -  114
Citations -  5512

Hubert Mönnikes is an academic researcher from Charité. The author has contributed to research in topics: Irritable bowel syndrome & Hypothalamus. The author has an hindex of 39, co-authored 109 publications receiving 5061 citations. Previous affiliations of Hubert Mönnikes include Humboldt University of Berlin & Humboldt State University.

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Intestinal fluid volumes and transit of dosage forms as assessed by magnetic resonance imaging

TL;DR: The gastrointestinal transit of sequentially administered capsules was investigated in relation to the availability of fluid along the intestinal lumen by magnetic resonance imaging.
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Role of stress in functional gastrointestinal disorders. Evidence for stress-induced alterations in gastrointestinal motility and sensitivity.

TL;DR: In experimental animals the most consistent pattern of GI motor alterations induced by various psychological and physical stressors is that of delaying gastric emptying and accelerating colonic transit, which suggests that stress affects visceral sensitivity in humans.
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Central nesfatin-1 reduces dark-phase food intake and gastric emptying in rats: differential role of corticotropin-releasing factor2 receptor.

TL;DR: Data indicate that nesfatin-1 acts centrally to reduce dark phase FI through CRF(2)-receptor-dependent pathways after forebrain injection and CRF (2) receptor-independent pathways after hindbrain injection.
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Somatic comorbidities of irritable bowel syndrome: A systematic analysis

TL;DR: The knowledge and structured assessment of comorbid somatic symptoms might allow to identify subgroups of IBS patients with special characteristics and lead to adaptation of the therapeutic concept.
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Role of CRF in stress-related alterations of gastric and colonic motor function.

TL;DR: The demonstration that central CRF plays a role in mediating gastric stasis resulting from surgery, peritonitis or high levels of central interleukin-1 provides new insight into the mechanisms involved in gastric ileus induced postoperatively or by infectious disease.