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James H. Dauber

Researcher at University of Pittsburgh

Publications -  156
Citations -  8000

James H. Dauber is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Transplantation & Lung transplantation. The author has an hindex of 51, co-authored 156 publications receiving 7741 citations. Previous affiliations of James H. Dauber include University of Pennsylvania.

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MMP1 and MMP7 as Potential Peripheral Blood Biomarkers in Idiopathic Pulmonary Fibrosis

TL;DR: The experiments provide the first evidence for a peripheral blood protein signature in IPF and the two main components, MMP7 and MMP1, are overexpressed in the lung microenvironment and distinguish IPF from other chronic lung diseases.
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Accelerated variant of idiopathic pulmonary fibrosis: clinical behavior and gene expression pattern.

TL;DR: A subgroup of IPF patients, predominantly smoking males, display an accelerated clinical course and have a gene expression pattern that is different from those with slower progression and longer survival, which may explain the difficulty in obtaining significant and reproducible results in studies of therapeutic interventions in patients with IPF.
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Carbon monoxide induces cytoprotection in rat orthotopic lung transplantation via anti-inflammatory and anti-apoptotic effects.

TL;DR: Data suggest that the anti-inflammatory and antiapoptotic properties of CO confer potent cytoprotection in a rat model of lung transplantation, which is limited by the high incidence of acute graft rejection.
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Influenza virus infection in adult solid organ transplant recipients.

TL;DR: Solid organ transplant recipients have been reported to be more susceptible to influenza virus, but little is known about the clinical epidemiology and the implications of influenza viral infection among SOT recipients.
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Immunologic Abnormalities in Sarcoidosis

TL;DR: Findings suggest that the lung is the site of an immune inflammatory response when involved in sarcoidosis, and many humoral abnormalities (for example, immune complexes) are absent in patients with resolved disease.