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James K. Weick

Researcher at Cleveland Clinic

Publications -  67
Citations -  4166

James K. Weick is an academic researcher from Cleveland Clinic. The author has contributed to research in topics: Chemotherapy & Vincristine. The author has an hindex of 29, co-authored 66 publications receiving 4103 citations. Previous affiliations of James K. Weick include Fred Hutchinson Cancer Research Center.

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Frequency and Clinical Significance of the Expression of the Multidrug Resistance Proteins MDR1/P-Glycoprotein, MRP1, and LRP in Acute Myeloid Leukemia. A Southwest Oncology Group Study

TL;DR: MDR1 is less frequent in younger AML patients, which may in part explain their better response to therapy and neither MRP1 nor LRP are significant predictors of outcome in this patient group, so inclusion of MDR1-modulators alone may benefit younger AMl patients with MDR 1(+) disease.
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A randomized investigation of high-dose versus standard-dose cytosine arabinoside with daunorubicin in patients with previously untreated acute myeloid leukemia: a Southwest Oncology Group study

TL;DR: Patients who received both HDAC induction and consolidation had the best postremission outcomes; however, the proportion of CR patients who did not go on to protocol consolidation therapy was more than twice as high afterHDAC induction compared with SDAC.
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Prognostic significance of Bcl-6 protein expression in DLBCL treated with CHOP or R-CHOP: a prospective correlative study.

TL;DR: A reduction in treatment failures and death with the addition of R to CHOP in Bcl-6(-) DLBCL cases only is observed, and the finding that Bcl(+) cases did not benefit from the additionof R toCHOP requires independent confirmation.
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N-ras mutations in adult de novo acute myelogenous leukemia: prevalence and clinical significance

TL;DR: A comparison of patients with and without N-ras mutations showed no statistically significant differences in pretreatment clinical variables, response to induction therapy, or survival, except for a possibly higher percentage of FAB M4 subtypes in patients with the N-ra mutation.