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Jean-Claude Willer

Researcher at French Institute of Health and Medical Research

Publications -  61
Citations -  4418

Jean-Claude Willer is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Reflex & Diffuse noxious inhibitory control. The author has an hindex of 31, co-authored 61 publications receiving 4251 citations. Previous affiliations of Jean-Claude Willer include Pierre-and-Marie-Curie University.

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Journal ArticleDOI

Psychophysical and electrophysiological approaches to the pain-relieving effects of heterotopic nociceptive stimuli.

Jean-Claude Willer, +2 more
- 01 Dec 1984 - 
TL;DR: The nociceptive flexion reflex (RIII reflex) and the concurrent subjective pain score elicited by right sural nerve stimulation at random intensities were studied in 10 healthy volunteers and a close relationship was found between the recruitment curves of the reflex and the pain score as a function of stimulus intensity.
Journal ArticleDOI

Restoration of normal motor control in Parkinson's disease during REM sleep

TL;DR: The restored motor control during REM sleep suggests a transient 'levodopa-like' reestablishment of the basal ganglia loop and parkinsonism may disappear by REM sleep-related disjunction between pyramidal and extrapyramidal systems.
Journal Article

Diffuse noxious inhibitory controls (DNIC) in animals and in man.

TL;DR: In this article, it was shown that lesions of the following structures did not modify DNIC: Periaqueductal grey (PAG), Cuneiform nucleus, Parabrachial area, locus coeruleus/subcoeruleus, rostral ventromedial medulla (RVM), including Raphe Magnus, Gigantocellularis and Paragigantocelluleis nuclei.
Book ChapterDOI

Diffuse Noxious Inhibitory Controls (DNIC)

TL;DR: This chapter summarizes experiments in both human beings and animals which show that diffuse noxious inhibitory controls (DNICs) are sustained by a well-defined neurological substratum based on spino-reticulo-spinal processes.
Journal ArticleDOI

Electromyography guides toward subgroups of mutations in muscle channelopathies

TL;DR: It is hypothesized that mutations are segregated into the different electromyographic patterns according to the underlying pathophysiological mechanisms and may be used in clinical practice as guides for molecular diagnosis of myotonia or periodic paralysis.