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Jelena Markovic

Researcher at University of Belgrade

Publications -  63
Citations -  1914

Jelena Markovic is an academic researcher from University of Belgrade. The author has contributed to research in topics: Sorption & Glutathione. The author has an hindex of 20, co-authored 59 publications receiving 1646 citations. Previous affiliations of Jelena Markovic include University of Valencia & University of British Columbia.

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A nuclear glutathione cycle within the cell cycle

TL;DR: A high level of GSH in the nucleus may not only have an immediate effect on gene expression patterns, but also contribute to how cells retain a memory of the cellular redox environment that is transferred through generations.
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Glutathione Is Recruited into the Nucleus in Early Phases of Cell Proliferation

TL;DR: It is reported here that cells concentrate GSH in the nucleus in the early phases of cell growth, when most of the cells are in an active division phase, and that GSH redistributes uniformly between the nucleus and the cytoplasm when cells reach confluence.
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Recruitment of glutathione into the nucleus during cell proliferation adjusts whole‐cell redox homeostasis in Arabidopsis thaliana and lowers the oxidative defence shield

TL;DR: In this paper, the authors show that GSH co-localizes with nuclear DNA during the proliferation of A.thaliana cells in culture and demonstrate that the GSH localization in the nucleus was observed in dividing pericycle cells of the lateral root meristem.
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Role of nuclear glutathione as a key regulator of cell proliferation.

TL;DR: The information provided in the present review suggests an important role of nuclear glutathione as a key regulator of epigenetic events that may be critical in the regulation of cell proliferation.
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Tuning to the significant: Neural and genetic processes underlying affective enhancement of visual perception and memory

TL;DR: The Biased Attention via Norepinephrine (BANE) model is presented, which unifies genetic, neuromodulatory, neural and behavioural evidence to account for ABA and examines differences arising from a variant of the ADRA2b gene.