J
Jennifer L. Moran
Researcher at Broad Institute
Publications - 119
Citations - 41227
Jennifer L. Moran is an academic researcher from Broad Institute. The author has contributed to research in topics: Genome-wide association study & Copy-number variation. The author has an hindex of 54, co-authored 114 publications receiving 33978 citations. Previous affiliations of Jennifer L. Moran include Scott & White Hospital & University of Illinois at Chicago.
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Journal ArticleDOI
A high incidence of Shigella -induced arthritis in a primate species: major histocompatibility complex class I molecules associated with resistance and susceptiblity, and their relationship to HLA-B27
Julie A. Urvater,Stephen McAdam,J. H. Loehrke,Todd M. Allen,Jennifer L. Moran,Thomas J. Rowell,S. Rojo,J A López de Castro,Joel D. Taurog,David I. Watkins +9 more
TL;DR: It is indicated that reactive arthritis can develop in this animal model in the absence of an HLA-B27-like molecule, and there was a statistically significant association between the rhesus macaque MHC A locus allele, Mamu-A*12, and the absenceof reactive arthritis following Shigella infection.
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A family-based study of common polygenic variation and risk of schizophrenia
Douglas M. Ruderfer,Douglas M. Ruderfer,George Kirov,Kimberly Chambert,Jennifer L. Moran,Michael John Owen,Michael Conlon O'Donovan,Pamela Sklar,Pamela Sklar,Shaun Purcell,Shaun Purcell +10 more
TL;DR: Whether putative risk alleles from a case/control study tend to be over-transmitted to offspring with schizophrenia is asked, to exclude bias due to cryptic population stratification as a possible source of inflated type I error.
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Genomic structure, mapping, and expression analysis of the mammalian Lunatic, Manic, and Radical fringe genes.
Jennifer L. Moran,Stuart H. Johnston,Cordelia Rauskolb,Jayant Bhalerao,Anne M. Bowcock,Thomas F. Vogt +5 more
TL;DR: Comparative analysis of mammalian Fringe genomic organization suggests that the first exon is evolutionarily labile and that the Fringe genes have a genomic structure distinct from those of previously characterized glycosyltransferases.
Journal ArticleDOI
Common alleles contribute to schizophrenia in CNV carriers [Erratum]
Katherine E. Tansey,Elliott Rees,David Edmun Johannes Linden,Stephan Ripke,Kimberly Chambert,Jennifer L. Moran,Steve McCarroll,Peter Holmans,George Kirov,James T.R. Walters,Michael John Owen,Michael Conlon O'Donovan +11 more
TL;DR: The first author in Reference 33 was listed incorrectly in the reference list and in the last paragraph of the Discussion section.
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Detecting large copy number variants using exome genotyping arrays in a large Swedish schizophrenia sample.
Jin P. Szatkiewicz,Benjamin M. Neale,Benjamin M. Neale,Colm O'Dushlaine,Menachem Fromer,Menachem Fromer,Jacqueline I. Goldstein,Jennifer L. Moran,Kimberly Chambert,Anna K. Kähler,Patrik K. E. Magnusson,Christina M. Hultman,Pamela Sklar,Shaun Purcell,Shaun Purcell,Steven A. McCarroll,Steven A. McCarroll,Patrick F. Sullivan,Patrick F. Sullivan +18 more
TL;DR: The results suggest the utility of exome-focused arrays in surveying large genic CNVs in very large samples; and open the door for new opportunities such as conducting well-powered CNV assessment and comparisons between different diseases.