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Jeny R. Cursino-Santos

Researcher at University of São Paulo

Publications -  9
Citations -  1290

Jeny R. Cursino-Santos is an academic researcher from University of São Paulo. The author has contributed to research in topics: Trichophyton rubrum & Gene. The author has an hindex of 6, co-authored 9 publications receiving 1225 citations.

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Comparison of the genomes of two Xanthomonas pathogens with differing host specificities

A.C.R. da Silva, +65 more
TL;DR: The genus Xanthomonas is a diverse and economically important group of bacterial phytopathogens, belonging to the γ-subdivision of the Proteobacteria, and several groups of strain-specific genes are identified and proposed mechanisms that may explain the differing host specificities and pathogenic processes are proposed.
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Transcriptional profiling reveals the expression of novel genes in response to various stimuli in the human dermatophyte Trichophyton rubrum.

TL;DR: This study identified novel T. rubrum genes that will be useful for ORF prediction in genome sequencing and facilitating functional genome analysis and challenging T.rubrum with cytotoxic drugs and ambient pH shifts extended the understanding of the molecular events possibly involved in the infectious process and resistance to antifungal drugs.
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Analysis of Trichophyton rubrum gene expression in response to cytotoxic drugs

TL;DR: In this paper, suppressive subtractive hybridization was used to isolate transcripts specifically upregulated during Trichophyton rubrum exposure to acriflavin, fluconazole, griseofulvin, terbinafine or undecanoic acid.
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Screening of filamentous fungi for lipase production: Hypocrea pseudokoningii a new producer with a high biotechnological potential

TL;DR: Filamentous fungi isolated from soil samples were screened for extracellular lipase production and the best producer was Hypocrea pseudokoningii identified by taxonomical criteria, and by rDNA sequencing of the variable internal transcribed spacers and the intervening 5.8S gene.
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Evolution of hepatitis C virus infection under host factor influence in an ethnically complex population.

TL;DR: The ethnic influence makes it difficult to reach a consensual definition of host‐dependent genetic factors controlling the hepatitis C virus (HCV) disease course.