scispace - formally typeset
J

Jerzy Stachura

Researcher at Jagiellonian University

Publications -  203
Citations -  5542

Jerzy Stachura is an academic researcher from Jagiellonian University. The author has contributed to research in topics: Gastric mucosa & Stomach. The author has an hindex of 40, co-authored 203 publications receiving 5431 citations. Previous affiliations of Jerzy Stachura include University of Tokyo & University of Göttingen.

Papers
More filters
Journal ArticleDOI

Prostaglandin protection of the gastric mucosa against alcohol injury--a dynamic time-related process. Role of the mucosal proliferative zone.

Andrzej S. Tarnawski, +4 more
- 01 Jan 1985 - 
TL;DR: The mucosal proliferative zone was severely damaged in controls within the first hour after ethanol administration, whereas 16,16-dimethyl prostaglandin E2 protected the zone from damage and prompt restoration of the surface epithelium and resumption of its barrier and transport functions.
Journal ArticleDOI

Inhibition of nitric oxide synthase delays healing of chronic gastric ulcers

Stanislaw J. Konturek, +4 more
- 03 Aug 1993 - 
TL;DR: It is concluded that endogenous NO plays an important role in the maintenance of blood flow around the ulcer, in the angiogenesis in the granulation tissue and in the healing of gastric ulcers.
Journal ArticleDOI

Prostaglandin Protection of Carbon Tetrachloride-Induced Liver Cell Necrosis in the Rat

Jerzy Stachura, +6 more
- 01 Nov 1981 - 
TL;DR: It is concluded that dmPGE2 protects the liver against cell necrosis induced by CCl4 in the rat.
Journal ArticleDOI

Increased expression of epidermal growth factor receptor during gastric ulcer healing in rats.

Andrzej S. Tarnawski, +4 more
- 01 Feb 1992 - 
TL;DR: An increased EGFR expression indicates an important role of EGF in ulcer healing and scar formation.
Journal ArticleDOI

Clinicopathologic profile of gastrointestinal stromal tumors (GISTs) with primary KIT exon 13 or exon 17 mutations: A multicenter study on 54 cases

Jerzy Lasota, +15 more
- 01 Apr 2008 - 
TL;DR: Evaluating the clinicopathologic profile of GISTs that have KIT exon 13 or exon 17 mutations found that Gastric KITExon 13 mutant GIST's tend to be slightly larger and more aggressive than gastric GIST’s in average, whereas the behavior of small intestinal Gists with KITexon 13 mutations does not differ from other small intestinalGISTs.