J
Jin Zhong
Researcher at Chinese Academy of Sciences
Publications - 172
Citations - 7453
Jin Zhong is an academic researcher from Chinese Academy of Sciences. The author has contributed to research in topics: Hepatitis C virus & Virus. The author has an hindex of 34, co-authored 149 publications receiving 6352 citations. Previous affiliations of Jin Zhong include Scripps Research Institute & ShanghaiTech University.
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Restoration of Bioactive Lantibiotic Suicin from a Remnant lan Locus of Pathogenic Streptococcus suis Serotype 2
TL;DR: A novel lantibiotic suicin was obtained by restoring its production from the remnant sui locus and demonstrated that virulence-associated SuiK-SuiR regulates its production.
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Molecular beam epitaxial growth, characterization and performance of high-detectivity GaInAsSb/GaSb PIN detectors operating at 2.0 to 2.6 μm
TL;DR: In this article, a PIN mesa front-illuminated photodetector with a maximum external quantum efficiency of 65% without anti-reflection coating and a peak detectivity D λ ∗ at 25 μm of 30 × 10 9 cm Hz 1/2 / W with a cut-off wavelength of 26 μm at room temperature have been achieved.
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Factors associated with spontaneous clearance of hepatitis C virus in Chinese population.
Fei Kong,Yu Pan,Xiumei Chi,Xiaomei Wang,Lin-Jiao Chen,Juan Lv,Haibo Sun,Ruihong Wu,Jinglan Jin,Ge Yu,Zhenhua Ma,Yang Wang,Xinxing Huang,Hua Li,Yang Bai,Jing Jia,Gerald Y. Minuk,Jin Zhong,Bing Sun,Jing Jiang,Junqi Niu +20 more
TL;DR: Spontaneous HCV clearance is more likely to occur in females, subjects with a history of icteric hepatitis, HBV coinfections, and those with the rs12979860 CC genotype.
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Production of hepatitis C virus lacking the envelope-encoding genes for single-cycle infection by providing homologous envelope proteins or vesicular stomatitis virus glycoproteins in trans.
TL;DR: Interestingly, it was found that vesicular stomatitis virus (VSV) glycoproteins could efficiently rescue the production of HCV lacking endogenous envelope proteins, which no longer required apolipoprotein E for virus production.
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Tuberous Sclerosis Complex Protein 2-Independent Activation of mTORC1 by Human Cytomegalovirus pUL38
TL;DR: This study showed that a mutant pUL38 lacking the N-terminal 24 amino acids (pHA-UL3825–331) was fully functional in suppressing cell death during infection and demonstrated that mTORC1 was activated by HCMV protein pul38 in both T SC2-dependent and TSC2-independent manners.