scispace - formally typeset
J

John J. Ohab

Researcher at University of California, Los Angeles

Publications -  6
Citations -  1672

John J. Ohab is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Neurogenesis & Subventricular zone. The author has an hindex of 5, co-authored 6 publications receiving 1583 citations. Previous affiliations of John J. Ohab include University of California, Santa Barbara.

Papers
More filters
Journal ArticleDOI

A Neurovascular Niche for Neurogenesis after Stroke

TL;DR: A novel brain environment for neuronal regeneration after stroke is defined and molecular mechanisms that are shared between angiogenesis and neurogenesis during functional recovery from brain injury are identified.
Journal ArticleDOI

A critical role of erythropoietin receptor in neurogenesis and post-stroke recovery.

TL;DR: The results suggest that both EPO and EPOR are essential for early embryonic neural development and that the classical EPOR is important for adult neurogenesis and for migration of regenerating neurons during post-injury recovery.
Journal ArticleDOI

Pten Deletion in Adult Neural Stem/Progenitor Cells Enhances Constitutive Neurogenesis

TL;DR: The data suggest that manipulating PTEN-controlled signaling pathways may be a useful step in facilitating endogenous neural stem/progenitor expansion for the treatment of disorders or lesions in regions associated with constitutive neurogenesis.
Journal ArticleDOI

Poststroke Neurogenesis: Emerging Principles of Migration and Localization of Immature Neurons

TL;DR: Most data on poststroke Neurogenesis derive from laboratory rodents, which may have an abnormal or blunted degree of neurogenesis and neuroplasticity compared to normal, wild rodents, and this will likely affect translational application of the principles of poststroke neuroGenesis from mouse to man.
Journal ArticleDOI

Determinants of the exclusion zone in dopaminergic amacrine cell mosaics.

TL;DR: Using nearest‐neighbor and Vornoi domain analysis, it is found that the dopaminergic amacrine cells were neither randomly distributed, nor did they achieve the regularity documented for other retinal cell types.