J
Jonathan G. Schoenecker
Researcher at Vanderbilt University Medical Center
Publications - 132
Citations - 2221
Jonathan G. Schoenecker is an academic researcher from Vanderbilt University Medical Center. The author has contributed to research in topics: Medicine & Thrombin. The author has an hindex of 24, co-authored 114 publications receiving 1618 citations. Previous affiliations of Jonathan G. Schoenecker include Boston Children's Hospital & Monroe Carell Jr. Children's Hospital at Vanderbilt.
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Journal ArticleDOI
The multifaceted role of fibrinogen in tissue injury and inflammation
TL;DR: There remains a critical need to define the precise temporal and spatial mechanisms by which fibrinogen-directed inflammatory events may dictate the severity of tissue injury and coordinate the remodeling and repair events essential to restore normal organ function.
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Complications and outcomes of diaphyseal forearm fracture intramedullary nailing: a comparison of pediatric and adolescent age groups.
Jeffrey E. Martus,Ryan K. Preston,Jonathan G. Schoenecker,Steven A. Lovejoy,Neil E. Green,Gregory A. Mencio +5 more
TL;DR: Flexible intramedullary nailing is an effective technique for pediatric forearm fractures with good to excellent outcomes in 91%.
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Micro-computed tomography assessment of the progression of fracture healing in mice
Kevin R. O'Neill,Christopher M. Stutz,Nicholas A. Mignemi,Michael C. Burns,Matthew R. Murry,Jeffry S. Nyman,Jeffry S. Nyman,Jonathan G. Schoenecker +7 more
TL;DR: Sample size estimates indicate that utilization of μCT requires fewer animals than biomechanics and thus is more practical for evaluating the healing femur in the mouse fracture model.
Journal ArticleDOI
Protease-activated receptor-2 signaling triggers dendritic cell development.
Ryan C. Fields,Jonathan G. Schoenecker,Justin P. Hart,Maureane Hoffman,Salvatore V. Pizzo,Jeffrey H. Lawson +5 more
TL;DR: It is shown that a serine protease acting via protease-activated receptor-2 (PAR-2) stimulates the development of DC from bone marrow progenitor cells cultured in granulocyte-macrophage colony-stimulating factor and IL-4.
Journal ArticleDOI
Bisphosphonates Inhibit Osteosarcoma-Mediated Osteolysis Via Attenuation of Tumor Expression of MCP-1 and RANKL
Tetsuro Ohba,Heather A. Cole,Justin M M Cates,David Slosky,Hirotaka Haro,Takashi Ando,Herbert S. Schwartz,Jonathan G. Schoenecker +7 more
TL;DR: It is established that osteosarcoma growth directly correlates with tumor‐induced osteolysis and activation of osteoclasts in vivo, and ZOL administration directly attenuates osteosARcoma production of RANKL/MCP‐1, reducing tumor‐ induced bone destruction.