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José Luis Zamorano

Researcher at Carlos III Health Institute

Publications -  740
Citations -  154643

José Luis Zamorano is an academic researcher from Carlos III Health Institute. The author has contributed to research in topics: Heart failure & Myocardial infarction. The author has an hindex of 105, co-authored 695 publications receiving 133396 citations. Previous affiliations of José Luis Zamorano include Hospital Clínico San Carlos & Universidad Francisco de Vitoria.

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Updated Clinical Practice Guidelines on Heart Failure: An International Alignment

TL;DR: The introduction of an angiotensin receptor-neprilysin inhibitor (ARNI), valsartan/sacubitril, and a sinoatrial node modulator, ivabradine, when applied judiciously, complement established pharmacological and device-based therapies, representing milestones in the evolution of care for patients with heart failure.
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Calcified right ventricular thrombus and antiphospholipid syndrome

TL;DR: A case about a 30-year-old woman, admitted with a 6-month history of arthralgia, fatigue, and intermittent fever, and the presence of a large and calcified mass in the right ventricular outflow tract attached to the subvalvular tricuspid apparatus is described.
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Hipertrofia ventricular izquierda por electrocardiograma o ecocardiograma y complicaciones cardiovasculares en hipertensos tratados de la Comunidad Autónoma de Madrid. Estudio MAVI-HTA*

TL;DR: Determinados pacientes hipertensos podrian beneficiarse del ecocardiograma en the valoracion del riesgo cardiovascular, y esta se asocio a una prevalencia of ECV tres veces superior.
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Effects of Ivabradine on Heart Rate and Hemodynamic Parameters in a Swine Model of Cardiogenic Shock.

TL;DR: Preliminary data indicate that ivabradine may offer a benefit in situations of severe tachycardia and shock, probably as a result of lower oxygen consumption and oxidative stress, although the hemodynamic effects of the drug in this context are unknown.
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Nuclear magnetic resonance spectroscopy of excised human hearts.

TL;DR: The results suggest that theosphocreatine concentration is lower in ischaemic heart disease than in dilated cardiomyopathy and that the phosphodiester peak is probably not useful in distinguishing between these two types of heart disease.