K
Karl Bauer
Researcher at Max Planck Society
Publications - 80
Citations - 4077
Karl Bauer is an academic researcher from Max Planck Society. The author has contributed to research in topics: Anterior pituitary & Thyrotropin-releasing hormone. The author has an hindex of 40, co-authored 80 publications receiving 3896 citations. Previous affiliations of Karl Bauer include Leibniz Association & Trinity College, Dublin.
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Journal ArticleDOI
Abnormal thyroid hormone metabolism in mice lacking the monocarboxylate transporter 8
Marija Trajkovic,Theo J. Visser,Jens Mittag,Sigrun Horn,Jan Lukas,Veerle Darras,Genadij Raivich,Karl Bauer,Heike Heuer +8 more
TL;DR: The circulating thyroid hormone levels of M CT8-null mice closely resemble those of humans with MCT8 mutations, yet in the mice, CNS development is only partially affected, indicating the hypothyroid state of this tissue.
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The Monocarboxylate Transporter 8 Linked to Human Psychomotor Retardation Is Highly Expressed in Thyroid Hormone-Sensitive Neuron Populations
Heike Heuer,Michael K. Maier,Sandra Iden,Jens Mittag,Edith C. H. Friesema,Theo J. Visser,Karl Bauer +6 more
TL;DR: Results strongly indicate that MCT8 plays an important role for proper central nervous system development by transporting TH into neurons as its main target cells.
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Folliculostellate cell network: a route for long-distance communication in the anterior pituitary.
TL;DR: Evidence is provided for an intrapituitary communication system by which information is transferred via the network of nonendocrine folliculostellate (FS) cells, which may provide an efficient mechanism that orchestrates anterior pituitary functioning in response to physiological needs.
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Expression of thyrotropin-releasing hormone receptor 2 (TRH-R2) in the central nervous system of rats.
TL;DR: The distribution ofTRH‐R2 mRNA seems to provide the anatomical basis for the described effects of TRH on higher cognitive functions as well as its effect on arousal, locomotor activity, and pain perception.
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Congenital hypothyroid female pax8-deficient mice are infertile despite thyroid hormone replacement therapy.
TL;DR: It is reported here that thyroxine-substituted female Pax8(-/-) mice are infertile because they lack a functional uterus revealing only remnants of myometrial tissue, pointing to a direct morphogenic role for Pax8 in uterine development.