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Katherine Halievski

Researcher at Michigan State University

Publications -  18
Citations -  413

Katherine Halievski is an academic researcher from Michigan State University. The author has contributed to research in topics: Atrophy & Neurotrophic factors. The author has an hindex of 9, co-authored 15 publications receiving 293 citations. Previous affiliations of Katherine Halievski include University of Toronto.

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Microglial P2X4R-evoked pain hypersensitivity is sexually dimorphic in rats.

TL;DR: The findings demonstrate the existence of sexually dimorphic pain signalling in rats, suggesting that this sex difference is evolutionarily conserved, at least across rodent species.
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Muscle Contraction Regulates BDNF/TrkB Signaling to Modulate Synaptic Function through Presynaptic cPKCα and cPKCβI

TL;DR: Results show that nerve-induced muscle contraction is a key regulator of BDNF/TrkB signaling pathway, retrogradely activating presynaptic cPKC isoforms (in particular cP KCβI) to modulate synaptic function, and indicates that a decrease in neuromuscular activity could affect the BDNF-TrkB/PKC pathway that links pre- and postsynaptic activity to maintain neuromUScular function.
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Sex-Dependent Mechanisms of Chronic Pain: A Focus on Microglia and P2X4R.

TL;DR: This minireview focuses on the roles of microglia and P2X4R in chronic pain, with specific attention to the circumstances under which these pathomechanisms differ between males and females.
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The effects of vitamin D3 during pregnancy and lactation on offspring physiology and behavior in Sprague–Dawley rats

TL;DR: In juveniles but not adults, variations in vitamin D were related to variations in measures of anxiety and marginally, activity levels, and both juveniles and adults were affected by mothers' diets.
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Decreased Peak Expiratory Flow Associated with Muscle Fiber-Type Switching in Spinal and Bulbar Muscular Atrophy.

TL;DR: In this paper, the authors compared the clinical and biochemical indices of spinal and bulbar muscular atrophy (SBMA) with those of amyotrophic lateral sclerosis (ALS) and found that %PEF is a disease-specific respiratory marker for the severity and progression of SBMA.