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Kevin J. Tracey

Researcher at The Feinstein Institute for Medical Research

Publications -  607
Citations -  90889

Kevin J. Tracey is an academic researcher from The Feinstein Institute for Medical Research. The author has contributed to research in topics: Inflammation & Cytokine. The author has an hindex of 138, co-authored 561 publications receiving 82791 citations. Previous affiliations of Kevin J. Tracey include Memorial Sloan Kettering Cancer Center & Hofstra University.

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Vagus nerve stimulation attenuates the systemic inflammatory response to endotoxin

TL;DR: Direct electrical stimulation of the peripheral vagus nerve in vivo during lethal endotoxaemia in rats inhibited TNF synthesis in liver, attenuated peak serum TNF amounts, and prevented the development of shock.
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HMG-1 as a Late Mediator of Endotoxin Lethality in Mice

TL;DR: High mobility group-1 (HMG-1) protein was found to be released by cultured macrophages more than 8 hours after stimulation with endotoxin, TNF, or IL-1, and showed increased serum levels after endotoxin exposure, suggesting that this protein warrants investigation as a therapeutic target.
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The inflammatory reflex

Kevin J. Tracey
- 19 Dec 2002 - 
TL;DR: The discovery that cholinergic neurons inhibit acute inflammation has qualitatively expanded understanding of how the nervous system modulates immune responses, and the opportunity now exists to apply this insight to the treatment of inflammation through selective and reversible 'hard-wired' neural systems.
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Nicotinic acetylcholine receptor alpha7 subunit is an essential regulator of inflammation.

TL;DR: It is reported that the nicotinic acetylcholine receptor α7 subunit is essential for inhibiting cytokine synthesis by the cholinergic anti-inflammatory pathway.
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Shock and tissue injury induced by recombinant human cachectin.

TL;DR: It appears that a single protein mediator (cachectin) is capable of inducing many of the deleterious effects of endotoxin.