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Kristine Phillips

Researcher at University of Michigan

Publications -  51
Citations -  3647

Kristine Phillips is an academic researcher from University of Michigan. The author has contributed to research in topics: Chronic pain & Arthritis. The author has an hindex of 29, co-authored 48 publications receiving 2966 citations. Previous affiliations of Kristine Phillips include Vanderbilt University Medical Center & Vanderbilt University.

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Mycophenolate mofetil versus oral cyclophosphamide in scleroderma-related interstitial lung disease (SLS II): a randomised controlled, double-blind, parallel group trial

TL;DR: In this article, the authors used a modified intention-to-treat analysis using an inferential joint model combining a mixed-effects model for longitudinal outcomes and a survival model to handle non-ignorable missing data.
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Myeloablative Autologous Stem-Cell Transplantation for Severe Scleroderma.

TL;DR: Myeloablative autologous hematopoietic stem‐cell transplantation achieved long‐term benefits in patients with scleroderma, including improved event‐free and overall survival, at a cost of increased expected toxicity.
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Central pain mechanisms in chronic pain states - Maybe it is all in their head

TL;DR: In chronic pain states, central nervous system factors appear to play particularly prominent roles, and central pain conditions respond best to CNS neuromodulating agents, such as serotonin-norepinephrine reuptake inhibitors (SNRIs) and anticonvulsants.
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Arthritis suppressor genes TIA-1 and TTP dampen the expression of tumor necrosis factor α, cyclooxygenase 2, and inflammatory arthritis

TL;DR: The results suggest that TIA-1 and TTP are genetic modifiers of inflammatory arthritis that can alter the spectrum of cells that produce arthritogenic cytokines.
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Central pain mechanisms in the rheumatic diseases: future directions.

TL;DR: There are very large inter-individual differences in these central nervous system factors that influence pain perception, such that some individuals with significant peripheral nociceptive input will feel little or no pain, whereas others are very pain sensitive, and they can experience pain with minimal or no identifiable abnormal peripheral nodal input.