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Lin Liu

Researcher at University of California, Riverside

Publications -  11
Citations -  962

Lin Liu is an academic researcher from University of California, Riverside. The author has contributed to research in topics: RNA silencing & Endoplasmic reticulum. The author has an hindex of 8, co-authored 9 publications receiving 765 citations. Previous affiliations of Lin Liu include Xinjiang University & Shenzhen University.

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Journal ArticleDOI

MicroRNAs Inhibit the Translation of Target mRNAs on the Endoplasmic Reticulum in Arabidopsis

TL;DR: It is shown that the translation inhibition, but not the mRNA cleavage activity, of Arabidopsis miRNAs requires ALTERED MERISTEM PROGRAM1 (AMP1), and AMP1-independent recruitment of miRNA target transcripts to membrane fractions shows that mi RNAs inhibit the translation of target RNAs on the ER.
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Oomycete pathogens encode RNA silencing suppressors

TL;DR: This work shows that two effectors from the oomycete plant pathogen Phytophthora sojae suppress RNA silencing in plants by inhibiting the biogenesis of small RNAs, identifying RNA silence suppression as a common strategy used by pathogens across kingdoms to cause disease.
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Biogenesis of phased siRNAs on membrane-bound polysomes in Arabidopsis

TL;DR: It is reported that Arabidopsis microRNAs and small interfering RNAs are distinctly partitioned between the endoplasmic reticulum (ER) and cytosol and the known functions of ribosomes and ER are expanded.
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RNA Quality Control as a Key to Suppressing RNA Silencing of Endogenous Genes in Plants

TL;DR: Recent advances on RNA quality control and its role in the suppression of RNA silencing at endogenous genes are reviewed and the mechanisms underlying the crosstalk among these pathways are discussed.
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The PROTEIN PHOSPHATASE4 Complex Promotes Transcription and Processing of Primary microRNAs in Arabidopsis.

TL;DR: R3A, together with one of two redundant catalytic subunit genes, PROTEIN PHOSPHATASE X (PPX)1 and PPX2, promotes the biogenesis of microRNAs (miRNAs) and hundreds of introns exhibit splicing defects in pp4r3a mutants are revealed.