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Louis H. Miller

Researcher at National Institutes of Health

Publications -  384
Citations -  36666

Louis H. Miller is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Plasmodium falciparum & Malaria. The author has an hindex of 101, co-authored 381 publications receiving 35104 citations. Previous affiliations of Louis H. Miller include Walter Reed Army Institute of Research & Guy's Hospital.

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Journal Article

International Union of Pharmacology. XXII. Nomenclature for Chemokine Receptors

TL;DR: A widely accepted receptor nomenclature system is described, ratified by the International Union of Pharmacology, that is facilitating clear communication in this area and updating current concepts of the biology and pharmacology of the chemokine system.
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Switches in expression of Plasmodium falciparum var genes correlate with changes in antigenic and cytoadherent phenotypes of infected erythrocytes.

TL;DR: It is shown that expression of variant antigenic determinants is correlated with expression of individual members of a large, multigene family named var, which is consistent with the involvement of var genes in antigenic variation and binding to endothelium.
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P. falciparum rosetting mediated by a parasite-variant erythrocyte membrane protein and complement-receptor 1

TL;DR: A new adhesive function for Pf EMP1 is described and the possibility that CR1 polymorphisms in Africans that influence the interaction between erythrocytes and PfEMP1 may protect against severe malaria is raised.
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Erythrocyte receptors for (Plasmodium knowlesi) malaria: Duffy blood group determinants

TL;DR: Duffy blood group negative human erythrocytes (FyFy) are resistant to infection by Plasmodium knowlesi, a simian malaria that infects Duffy positive human ERYthrocycles, which suggests that Duffy blood group determinants (Fya or Fyb) may be ery Throcyte receptors for P. vivax.
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A receptor for the malarial parasite Plasmodium vivax: the erythrocyte chemokine receptor

TL;DR: Evidence is indicated that the Duffy blood group antigen is the erythrocyte receptor for the chemokines interleukin-8 (IL-8) and melanoma growth stimulatory activity (MGSA) and the possibility of receptor blockade for anti-malarial therapy is suggested.