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Lucia Grasso

Researcher at University of Pisa

Publications -  113
Citations -  6807

Lucia Grasso is an academic researcher from University of Pisa. The author has contributed to research in topics: Thyroid & Thyroid cancer. The author has an hindex of 45, co-authored 113 publications receiving 6411 citations. Previous affiliations of Lucia Grasso include University of Molise & World Health Organization.

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Impact of Routine Measurement of Serum Calcitonin on the Diagnosis and Outcome of Medullary Thyroid Cancer: Experience in 10,864 Patients with Nodular Thyroid Disorders

TL;DR: This study confirms that MTC is not an infrequent finding among patients with thyroid nodules (nearly 1 in 250 patients) and allows the diagnosis and treatment of MTC at an earlier stage, resulting in a better outcome compared with MTC not detected by serum CT measurement.
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The spectrum of thyroid disorders in an iodine-deficient community: the Pescopagano survey.

TL;DR: In the present survey of an iodine-deficient community, a progressive increase with age of goiter prevalence, thyroid nodularity, and functional autonomy was observed, twice as high as that reported in iodine-sufficient areas, mainly due to an increased frequency of toxic nodular goiter.
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Recombinant Human Thyrotropin-Stimulated Serum Thyroglobulin Combined with Neck Ultrasonography Has the Highest Sensitivity in Monitoring Differentiated Thyroid Carcinoma

TL;DR: The rhTSH-stimulated Tg test combined with neck ultrasonography has the highest diagnostic accuracy in detecting persistent disease in the follow-up of differentiated thyroid carcinoma.
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Disappearance of humoral thyroid autoimmunity after complete removal of thyroid antigens.

TL;DR: A large group of patients with differentiated thyroid carcinoma who had serum thyroid peroxidase, thyroglobulin, or TSH-receptor antibodies due to coexistent thyroid autoimmune disease were studied, finding gradual disappearance of antibodies gradually disappeared in most patients.
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Mutations in the selenocysteine insertion sequence–binding protein 2 gene lead to a multisystem selenoprotein deficiency disorder in humans

TL;DR: Mutation of SECISBP2 is associated with a multisystem disorder with defective biosynthesis of many selenoproteins, highlighting their role in diverse biological processes.