M
Marc-Henri Stern
Researcher at Curie Institute
Publications - 196
Citations - 12240
Marc-Henri Stern is an academic researcher from Curie Institute. The author has contributed to research in topics: Gene & Breast cancer. The author has an hindex of 49, co-authored 168 publications receiving 9402 citations. Previous affiliations of Marc-Henri Stern include Necker-Enfants Malades Hospital & French Institute of Health and Medical Research.
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Genomic Amplification Is Not a Frequent Event in Uveal
TL;DR: In this article, the authors performed copy number and genotyping analysis on formalin-fixed, parafree-embedded (FFPE)samples of 58 cases with the genome-wide SNP arraysversion 6.0 (Affymetrix, Santa Clara, CA).
Journal ArticleDOI
Cytidine analogs are synthetic lethal with base excision repair default due to MBD4 deficiency
T. Chabot,Fariba Nemati,Aurélie Herbette,Alex Demeyer,Stéphane Dayot,Olivier Ganier,Samar Alsafadi,Sophie Gardrat,Pascale Mariani,Marie Luporsi,Maxime Corbé,Vincent Servois,Nathalie Cassoux,Didier Decaudin,Sergio Roman Roman,Elaine Del Nery,Sophie Piperno-Neumann,Marc-Henri Stern,Manuel Rodrigues +18 more
TL;DR: In this paper , the difference in DNA metabolism consecutive to MBD4 deficiency may result in specific cytotoxicities in MBD-4-deficient tumor cells in a synthetic lethality fashion.
Book ChapterDOI
Ataxie-télangiectasie et syndromes apparentés
Dominique Stoppa-Lyonnet,Marianne Debré,Romain Micol,Lilia Ben Slama,Jérême Couturier,Marc-Henri Stern +5 more
TL;DR: In this article, les caracteristiques cliniques, biologiques, and moleculaires de l'ataxia-telangiectasia mutated (A-T) are decrirons, ainsi que celles de deux pathologies proches qui ont en commun ses anomalies cytogenetiques : A-T like disorder (ATLD) and syndrome de Nijmegen.
genetic alteration in T-cell acute lymphoblastic leukemia Multiple tumor-suppressor gene 1 inactivation is the most frequent
TL;DR: The author’s website is www.hematologylibrary.org, which can be found online at http://bloodjournal.org/site/misc/rights.xhtml.
Journal ArticleDOI
DNA Replication Stress Due to Loss of R-Loops in Myelodysplastic Syndromes with SF3B1 Mutation
David Rombaut,Carine Lefevre,Batoul Farhat,Sabrina Bondu,Anne Letessier,Auriane Lesieur-Pasquier,Daisy Castillo-Guzman,Marjorie Leduc,Emilie-Fleur Gautier,Virginie Chesnais,Alice Rousseau,Ismael Boussaid,Sarah Battault,Alexandre Houy,Didier Bouscary,Lise Willems,Nicolas Chapuis,Sophie Park,Sophie Raynaud,Thomas Cluzeau,Emmanuelle Clappier,Pierre Fenaux,Lionel Ades,Eric Solary,Raphaël Margueron,Michel Wassef,Olivier Kosmider,Samar Alsafadi,Nathalie Droin,Angelos Constantinou,Marc-Henri Stern,Benoit Miotto,Frédéric Chédin,Michaela Fontenay +33 more
TL;DR: In this article , the authors performed RNA-seq analysis on myelodysplastic syndromes (MDS) with SF3B1 mutation and 43 other low risk MDS was enrolled in this study.