scispace - formally typeset
M

Marek Malik

Researcher at National Institutes of Health

Publications -  548
Citations -  63023

Marek Malik is an academic researcher from National Institutes of Health. The author has contributed to research in topics: QT interval & Myocardial infarction. The author has an hindex of 78, co-authored 535 publications receiving 58778 citations. Previous affiliations of Marek Malik include St. George's University & Imperial College London.

Papers
More filters
Journal ArticleDOI

Short- and long-term reproducibility of QT, QTc, and QT dispersion measurement in healthy subjects.

TL;DR: The study investigated interobserver and intrasubject reproducibility of QT interval duration and dispersion measured in standard 12‐lead ECGs recorded at 25 mm/sec and showed that the measurement of the QT intervals from standard ECG recordings is feasible and not operator dependent.
Journal ArticleDOI

Analysis of 12-Lead T-Wave Morphology for Risk Stratification After Myocardial Infarction

TL;DR: Computerized T-wave morphology analysis of the 12-lead resting ECG permits independent assessment of post-MI risk and an improved risk stratification when combined with other risk markers.
Journal ArticleDOI

Distinction between arrhythmic and nonarrhythmic death after acute myocardial infarction based on heart rate variability, signal-averaged electrocardiogram, ventricular arrhythmias and left ventricular ejection fraction.

TL;DR: Heart rate variability, the signal-averaged electrocardiogram (ECG), ventricular arrhythmias and left ventricular ejection fraction predict the mechanism of cardiac death after myocardial infarction and a combination of risk factors identified patient groups in which a majority of deaths were either arrhythmic or nonarrhythmmic.
Journal ArticleDOI

Baroreflex sensitivity and electrophysiological correlates in patients after acute myocardial infarction

TL;DR: This study confirms that depressed BRS identifies a subgroup at high risk for arrhythmic events after myocardial infarction and that programmed ventricular stimulation may be safely limited to this group without any loss of predictive accuracy.