M
Marina Babić
Researcher at University of Rijeka
Publications - 22
Citations - 1606
Marina Babić is an academic researcher from University of Rijeka. The author has contributed to research in topics: Receptor & Immune system. The author has an hindex of 16, co-authored 20 publications receiving 1386 citations. Previous affiliations of Marina Babić include Leibniz Association.
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Journal ArticleDOI
Peptide-specific recognition of human cytomegalovirus strains controls adaptive natural killer cells
Quirin Hammer,Timo Rückert,Eva Maria Borst,Josefine Dunst,André Haubner,Pawel Durek,Frederik Heinrich,Gilles Gasparoni,Marina Babić,Adriana Tomic,Gabriella Pietra,Mikalai Nienen,Igor Wolfgang Blau,Jörg Hofmann,Il-Kang Na,Immo Prinz,Christian Koenecke,P.G. Hemmati,Nina Babel,Nina Babel,Renate Arnold,Jörn Walter,Kevin Thurley,Mir-Farzin Mashreghi,Martin Messerle,Chiara Romagnani +25 more
TL;DR: It is proposed that polymorphic H CMV peptides contribute to shaping of the heterogeneity of adaptive NKG2C+ NK cell populations among HCMV-seropositive people and demonstrate adaptive-like recognition that can discriminate between closely related viral strains.
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Degradation of cellular mir-27 by a novel, highly abundant viral transcript is important for efficient virus replication in vivo.
Lisa Marcinowski,Mélanie Tanguy,Astrid Krmpotić,Bernd Rädle,Vanda Juranić Lisnić,Lee Tuddenham,Béatrice Chane-Woon-Ming,Zsolt Ruzsics,Florian Erhard,Corinna Benkartek,Marina Babić,Ralf Zimmer,Joanne Trgovcich,Ulrich H. Koszinowski,Stipan Jonjić,Sébastien Pfeffer,Lars Dölken,Lars Dölken +17 more
TL;DR: Three mutant viruses no longer able to target miR-27a/b, either due to miRNA target site disruption or target site replacement, showed significant attenuation in multiple organs as early as 4 days post infection, indicating that degradation of miR/b is important for efficient MCMV replication in vivo.
Journal ArticleDOI
Immune evasion of natural killer cells by viruses
TL;DR: Improving the understanding of viral regulation of NK cell function could be essential for designing more efficient measures in the prophylaxis and treatment of virus-induced pathology.
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RORγt+ Innate Lymphoid Cells Acquire a Proinflammatory Program upon Engagement of the Activating Receptor NKp44
Timor Glatzer,Monica Killig,Johannes Meisig,Johannes Meisig,Isabelle Ommert,Merlin Luetke-Eversloh,Marina Babić,Daniela Paclik,Nils Blüthgen,Nils Blüthgen,Rainer Seidl,C Seifarth,Jörn Gröne,Minoo Lenarz,K Stölzel,Dominik Fugmann,Angel Porgador,Anja E. Hauser,Alexander Karlas,Chiara Romagnani +19 more
TL;DR: It is shown that RORγt⁺ ILCs can directly sense the environment by the engagement of the activating receptor NKp44 and are able to switch between IL-22 or TNF production, depending on the triggering stimulus.
Journal ArticleDOI
Human RORγt+CD34+ Cells Are Lineage-Specified Progenitors of Group 3 RORγt+ Innate Lymphoid Cells
Elisa Montaldo,Luiz Gustavo Teixeira-Alves,Timor Glatzer,Pawel Durek,Ulrik Stervbo,Wiebke Hamann,Marina Babić,Daniela Paclik,K Stölzel,Jörn Gröne,Laura Lozza,Kerstin Juelke,Nadine Matzmohr,Fabrizio Loiacono,Francesca Petronelli,Nicholas D. Huntington,Lorenzo Moretta,Maria Cristina Mingari,Chiara Romagnani +18 more
TL;DR: It is demonstrated that in humans RORγt(+)CD34(+) cells are lineage-specified progenitors of IL-22(+) ILC3s and proposed that tonsils and intestinal LP, which are enriched both in committed precursors and mature I LC3s, might represent preferential sites of ILC2 lineage differentiation.