M
Mark M. Kockx
Researcher at University of Antwerp
Publications - 159
Citations - 12365
Mark M. Kockx is an academic researcher from University of Antwerp. The author has contributed to research in topics: Apoptosis & Macrophage. The author has an hindex of 55, co-authored 153 publications receiving 11076 citations.
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Journal ArticleDOI
Human granulosa cells after ovulation induction show caspase-independent cell death.
TL;DR: Support is provided for a caspase-independent cell death mechanism in human granulosa-lutein cells after ovulation induction during an in vitro fertilization procedure.
Journal ArticleDOI
Stromal marker fibroblast activation protein drives outcome in T1 non-muscle invasive bladder cancer.
Tim Muilwijk,Murat Akand,Sofie Daelemans,Koen M. Marien,Yannick Waumans,Mark M. Kockx,Loïc Baekelandt,Thomas Van den Broeck,Frank Van der Aa,Thomas Gevaert,Steven Joniau +10 more
TL;DR: Fibroblast activation protein-α (FAP) is a transmembrane peptidase and a surrogate marker for cancerassociated fibroblasts (CAFs) as mentioned in this paper.
Journal ArticleDOI
Digital pathology in immuno-oncology – a roadmap for clinical development
TL;DR: Implementing validation and quality assurance activities for digital pathology in oncology clinical development will play a vital role to establish standards and use cases, which may then serve as subsequent metrics for clinical consideration.
Book ChapterDOI
Inhibition of interleukin-1β convertase is associated with decrease of neointimal hyperplasia after coronary artery stenting in pigs
Mariann Gyöngyösi,Wolfgang Sperker,Csaba Csonka,Diana Bonderman,Irene Lang,Christoph Strehblow,Christopher Adlbrecht,Maryam Shirazi,Ursula Windberger,Stephan Marlovits,Michael Gottsauner-Wolf,Paul Wexberg,Mark M. Kockx,Péter Ferdinandy,Dietmar Glogar +14 more
TL;DR: In conclusion, intracoronary administration of Ac-YVAD-cmk before coronary artery stenting results in significantly decreased neointimal hyperplasia due to the inhibition of local IL-lβ production and decreased neointedimal apoptosis.
Journal ArticleDOI
The relationship between pre-existing subendothelial smooth muscle cell accumulations and foam cell lesions in cholesterol-fed rabbits
TL;DR: The newly formed FC plaques remained clearly separated from the pre-existing subendothelial SMC, which can be attributed not only to monocyte recruitment but also to macrophage multiplication.