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Marshall Devor

Researcher at Hebrew University of Jerusalem

Publications -  260
Citations -  21843

Marshall Devor is an academic researcher from Hebrew University of Jerusalem. The author has contributed to research in topics: Neuropathic pain & Nerve injury. The author has an hindex of 75, co-authored 253 publications receiving 20746 citations. Previous affiliations of Marshall Devor include University of Washington & McGill University.

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A putative flip–flop switch for control of REM sleep

TL;DR: A brainstem flip–flop switch is proposed, consisting of mutually inhibitory REM-off and REM-on areas in the mesopontine tegmentum that contain GABA (γ-aminobutyric acid)-ergic neurons that heavily innervate the other.
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Peripheral nerve injury triggers noradrenergic sprouting within dorsal root ganglia

TL;DR: It is reported that, after sciatic nerve ligation, noradrenergic perivascular axons in rats sprout into dorsal root ganglia and form basket-like structures around large-diameter axotomized sensory neurons; sympathetic stimulation can activate such neurons repetitively.
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Heritability of nociception I: responses of 11 inbred mouse strains on 12 measures of nociception.

TL;DR: This report begins to systematically describe and characterize genetic variability of nociception in a mammalian species, Mus musculus, using 11 readily-available inbred mouse strains and demonstrates the existence of clear strain differences in each assay.
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Autotomy following peripheral nerve lesions: experimental anaesthesia dolorosa.

TL;DR: The time course and degree of autotomy following various types of nerve injury in rats and mice is described and reasons are given to propose that autotomy is triggered by an abnormal afferent barrage generated in the cut end of the nerve.
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Sensory afferent impulses originate from dorsal root ganglia as well as from the periphery in normal and nerve injured rats

TL;DR: The dorsal root ganglion with its ongoing activity and mechanical sensitivity could be a source of pain producing impulses and could particularly contribute to pain in those conditions of peripheral nerve damage where pain persists after peripheral anaesthesia or where vertebral manipulation is painful.