M
Martin Fenner
Researcher at Hannover Medical School
Publications - 119
Citations - 3705
Martin Fenner is an academic researcher from Hannover Medical School. The author has contributed to research in topics: Scholarly communication & Computer science. The author has an hindex of 29, co-authored 105 publications receiving 3423 citations. Previous affiliations of Martin Fenner include Charité & PLOS.
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Journal ArticleDOI
European Consensus Conference on Diagnosis and Treatment of Germ Cell Cancer: A Report of the Second Meeting of the European Germ Cell Cancer Consensus group (EGCCCG): Part I
Susanne Krege,Jörg Beyer,Rainer Souchon,Peter Albers,Walter Albrecht,Ferran Algaba,Michael Bamberg,István Bodrogi,Carsten Bokemeyer,Eva Cavallin-Ståhl,Johannes Classen,C Clemm,Gabriella Cohn-Cedermark,Stéphane Culine,Gedske Daugaard,Pieter H.M. De Mulder,Maria De Santis,Maike de Wit,Ronald de Wit,H. G. Derigs,Klaus Peter Dieckmann,Annette Dieing,Jean Pierre Droz,Martin Fenner,Karim Fizazi,Aude Flechon,Sophie D. Fosså,Xavier Garcia del Muro,Thomas Gauler,Lajos Géczi,Arthur Gerl,Jose Ramon Germa-Lluch,Silke Gillessen,Jörg T. Hartmann,Michael Hartmann,Axel Heidenreich,Wolfgang Hoeltl,Alan Horwich,Robert Huddart,Michael Jewett,Johnathan Joffe,William G. Jones,László Kisbenedek,Olbjørn Klepp,S. Kliesch,Kai Uwe Koehrmann,Christian K. Kollmannsberger,Markus A. Kuczyk,Pilar Laguna,Oscar Leiva Galvis,Volker Loy,Malcolm David Mason,Graham M. Mead,Rolf Mueller,Craig R. Nichols,Nicola Nicolai,Tim Oliver,D. Ondruš,Gosse O N Oosterhof,Luis Paz Ares,Giorgio Pizzocaro,Jörg Pont,Tobias Pottek,Thomas Powles,Oliver Rick,Giovanni Rosti,Roberto Salvioni,Jutta Scheiderbauer,Hans U. Schmelz,Heinz Schmidberger,Hans-Joachim Schmoll,Mark Schrader,Felix Sedlmayer,Niels E. Skakkebæk,Aslam Sohaib,Sergei Tjulandin,Padraig Warde,Stefan Weinknecht,Lothar Weissbach,Christian Wittekind,Eva Winter,Lori Wood,Hans von der Maase +82 more
TL;DR: F refinements in the treatment of early- and advanced-stage testicular cancer have emerged from clinical trials, and expert clinical skills will continue to be one of the major determinants for the prognosis of patients with germ cell cancer.
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X inactivation in the mouse embryo deficient for Dnmt1: distinct effect of hypomethylation on imprinted and random X inactivation.
TL;DR: It is suggested that maintenance of imprinted X in activation in the extraembryonic lineage can tolerate extensive demethylation while normal levels of methylation are required for stable maintenance of X inactivation in the embryonic lineage.
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Maintaining success, reducing treatment burden, focusing on survivorship: highlights from the third European consensus conference on diagnosis and treatment of germ-cell cancer
Jörg Beyer,Peter Albers,Renske Altena,Jorge Aparicio,Carsten Bokemeyer,Jonas Busch,Richard Cathomas,Eva Cavallin-Ståhl,Noel W. Clarke,J. Claßen,Gabriella Cohn-Cedermark,Alv A. Dahl,Gedske Daugaard,U. De Giorgi,M. De Santis,M. de Wit,R. de Wit,Klaus-Peter Dieckmann,Martin Fenner,Karim Fizazi,Aude Flechon,Sophie D. Fosså,J. R. Germá Lluch,Jourik A. Gietema,Silke Gillessen,Aleksander Giwercman,J. T. Hartmann,Axel Heidenreich,Marcus Hentrich,Friedemann Honecker,Alan Horwich,Robert Huddart,S. Kliesch,Christian Kollmannsberger,Susanne Krege,M.P. Laguna,Leendert H. J. Looijenga,Anja Lorch,Jean-Pierre Lotz,Frank Mayer,Andrea Necchi,Nicola Nicolai,Janine Nuver,K. Oechsle,Jan Oldenburg,J. W. Oosterhuis,Thomas Powles,E. Rajpert-De Meyts,Oliver Rick,Giovanni Rosti,Roberto Salvioni,Mark Schrader,S. Schweyer,Felix Sedlmayer,Aslam Sohaib,Rainer Souchon,T. Tandstad,C. Winter,C. Wittekind +58 more
TL;DR: The main recommendations and controversies of this meeting are presented, with a particular focus on acute and late toxic effects as well as on survivorship issues.
Journal ArticleDOI
ORCID: a system to uniquely identify researchers
TL;DR: Use cases for embedding ORCID identifiers in manuscript submission workflows, prior work searches, manuscript citations, and repository deposition are described and recommendations for storing and displaying ORC ID identifiers in publication metadata are made.
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PPARγ Ligands and ATRA Inhibit the Invasion of Human Breast Cancer Cells in vitro
TL;DR: Treatment of the highly aggressive human breast cancer cell line MDA-MB-231 with the synthetic PPARγ ligands pioglitazone (PGZ), rosiglitazonesone (RGZ), GW7845 or its natural ligand 15-deoxy-Δ 12, 14-prostaglandin J2(15d-PGJ2), at concentrations at which no obvious cytotoxicity was observed in vitro, led to a significant inhibition of