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Martin Lévesque

Researcher at Laval University

Publications -  100
Citations -  5221

Martin Lévesque is an academic researcher from Laval University. The author has contributed to research in topics: Dopaminergic & Smart grid. The author has an hindex of 35, co-authored 96 publications receiving 4688 citations. Previous affiliations of Martin Lévesque include Université de Montréal & University of Pittsburgh.

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Newly generated neurons in the amygdala and adjoining cortex of adult primates

TL;DR: The production of newborn neurons in the amygdala, piriform cortex, and inferior temporal cortex seems to parallel the continuing addition of neurons inThe olfactory bulb, and these two concomitant phenomena may ensure structural stability and functional plasticity to the primate Olfactory system and temporal lobe.
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Elevated Mitochondrial Bioenergetics and Axonal Arborization Size Are Key Contributors to the Vulnerability of Dopamine Neurons.

TL;DR: This work shows for the first time that vulnerable nigral DA neurons differ from less vulnerable DA neurons by having a higher basal rate of mitochondrial OXPHOS, a smaller reserve capacity, a higher density of axonal mitochondria, an elevated level of basal oxidative stress, and a considerably more complex axonal arborization.
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Organization of the basal ganglia: the importance of axonal collateralization.

TL;DR: The basal ganglia now stand as a widely distributed neuronal network, whose elements are endowed with a highly patterned set of axon collaterals, and the elucidation of this finely tuned network is needed to understand the complex spatiotemporal sequence of neural events that ensures the flow of cortical information through the basal Ganglia.
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Single-axon tracing study of neurons of the external segment of the globus pallidus in primate.

TL;DR: The results suggest that the GPe is an important integrative locus in primate basal ganglia that allows single GPe neurons to exert a multifarious effect not only on the STN, which is the claimed GPe target, but also on the two major output structures of the basal Ganglia, the SNr and the GPi.
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Boc and Gas1 each form distinct Shh receptor complexes with Ptch1 and are required for Shh-mediated cell proliferation

TL;DR: It is shown that, unexpectedly, cerebellar granule neuron progenitors lacking Boc and Cdon, the vertebrate orthologs of Ihog and Boi, still proliferate in response to Hh, and a distinct requirement for ligand-binding components that distinguishes the vertebrates and invertebrate Hh receptor systems is revealed.