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Martin von Bergen

Researcher at Helmholtz Centre for Environmental Research - UFZ

Publications -  395
Citations -  19405

Martin von Bergen is an academic researcher from Helmholtz Centre for Environmental Research - UFZ. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 58, co-authored 346 publications receiving 15305 citations. Previous affiliations of Martin von Bergen include Max Planck Society & Leipzig University.

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Physiology of Geobacter metallireducens under excess and limitation of electron donors. Part I. Batch cultivation with excess of carbon sources.

TL;DR: G. metallireducens seems to lack strong carbon catabolite repression under high substrate concentrations, which might be advantageous for survival in habitats rich in fatty acids and aromatic hydrocarbons.
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Sulfated hyaluronan attenuates inflammatory signaling pathways in macrophages involving induction of antioxidants.

TL;DR: This study used global quantitative proteomics combined with targeted analysis of key proteins to characterize the effect of sL‐HA on fully differentiated human inflammatory macrophages (iMФ), indicating a profound counter‐regulation of their initial inflammatory phenotype.
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Glucocorticoid Treatment Leads to Aberrant Ion and Macromolecular Transport in Regenerating Zebrafish Fins.

TL;DR: The data indicate that continued synthetic glucocorticoid exposure in zebrafish deteriorates cellular trafficking processes in the regenerating fin, which interferes with appropriate tissue restoration upon injury.
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Functional analysis of an anaerobic m-xylene-degrading enrichment culture using protein-based stable isotope probing

TL;DR: The data suggest that m-xylene is assimilated by the Desulfobacteriaceae phylotype, whereas the role of the Epsilonproteobacterium in the consortium remained unclear.
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Proteome changes in human bronchoalveolar cells following styrene exposure indicate involvement of oxidative stress in the molecular-response mechanism.

TL;DR: The results underline the relevance of oxidative stress as a primary molecular response mechanism of lung epithelial cells to styrene exposure at indoor‐relevant concentrations.