M
Mary J. Roman
Researcher at Cornell University
Publications - 371
Citations - 51574
Mary J. Roman is an academic researcher from Cornell University. The author has contributed to research in topics: Blood pressure & Population. The author has an hindex of 100, co-authored 365 publications receiving 48687 citations. Previous affiliations of Mary J. Roman include University of Maryland, Baltimore & NewYork–Presbyterian Hospital.
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Journal ArticleDOI
Cardiac and Systemic Hemodynamic Characteristics of Hypertension and Prehypertension in Adolescents and Young Adults. The Strong Heart Study
Jennifer S. Drukteinis,Mary J. Roman,Richard R. Fabsitz,Elisa T. Lee,Lyle G. Best,Marie Russell,Richard B. Devereux +6 more
TL;DR: Despite the young age of participants with hypertension and prehypertension, they had prognostically adverse preclinical cardiovascular disease, including left ventricular hypertrophy and evidence of increased arterial stiffness.
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Relation of arterial structure and function to left ventricular geometric patterns in hypertensive adults
TL;DR: Among patients with generally mild, uncomplicated systemic hypertension, arterial structure and function are most abnormal when concentric left ventricular hypertrophy is present and may contribute to the more adverse outcome associated with this geometric pattern.
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Evaluation of Concentric Left Ventricular Geometry in Humans: Evidence for Age-Related Systematic Underestimation
Giovanni de Simone,Stephen R. Daniels,Thomas R. Kimball,Mary J. Roman,Carmela Romano,Marcello Chinali,Maurizio Galderisi,Richard B. Devereux +7 more
TL;DR: Prevalence of concentric LV hypertrophy is suggested to be defined by coexistence of high LV mass with age-normalized RWTm >0.41 or RWTp <0.40, as well as in hypertensive subjects.
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Genetic and Environmental Contributions to Cardiovascular Disease Risk in American Indians The Strong Heart Family Study
Kari E. North,Barbara V. Howard,Thomas K. Welty,Lyle G. Best,Elisa T. Lee,J. L. Yeh,Richard R. Fabsitz,Mary J. Roman,Jean W. MacCluer +8 more
TL;DR: The results suggest that heredity explains a substantial proportion of the variability of CVD risk factors and that these heritabilities are large enough to warrant a search for major risk factor genes.
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Identification of a Chromosome 11q23.2-q24 Locus for Familial Aortic Aneurysm Disease, a Genetically Heterogeneous Disorder
Carl J. Vaughan,Mairead Casey,Jie He,Mark Veugelers,Kiersten A. Henderson,Dongchuan Guo,Robert Campagna,Mary J. Roman,Dianna M. Milewicz,Richard B. Devereux,Craig T. Basson +10 more
TL;DR: FAA disease is genetically heterogeneous and a novel FAA locus is identified at chromosome 11q23.3-q24, a critical step toward elucidating 1 gene defect responsible for aortic dilatation, and future characterization of the FAA1 gene will enhance the ability to achieve presymptomatic diagnosis of aneurysms.