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Mary J. Roman

Researcher at Cornell University

Publications -  371
Citations -  51574

Mary J. Roman is an academic researcher from Cornell University. The author has contributed to research in topics: Blood pressure & Population. The author has an hindex of 100, co-authored 365 publications receiving 48687 citations. Previous affiliations of Mary J. Roman include University of Maryland, Baltimore & NewYork–Presbyterian Hospital.

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Left ventricular wall stresses and wall stress–mass–heart rate products in hypertensive patients with electrocardiographic left ventricular hypertrophy: the LIFE study

TL;DR: Hypertensive patients with ECG LV hypertrophy have increased LV wall stresses and stress-mass-heart rate products, suggesting a contribution of high myocardial oxygen demand to increased risk in such patients.
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Systemic Lupus Erythematosus Predicts Increased Left Ventricular Mass

TL;DR: SLE predicts increased LV mass, possibly because of inflammation-related arterial stiffening, and may contribute to the increased cardiac morbidity and mortality observed in SLE patients.
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Prevalence of hypertrophic cardiomyopathy in a population-based sample of American Indians aged 51 to 77 years (the Strong Heart Study).

TL;DR: Clinical unrecognized HC was present in 1:500 of an aging American Indian cohort, offering evidence that HC is a relatively common genetic disease with widespread occurrence within the United States.
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Association of the auscultatory gap with vascular disease in hypertensive patients

TL;DR: The goals were to determine the prevalence of the auscultatory gap in a large cohort of asymptomatic patients with hypertension who were not receiving medication and evaluate the relation between the aUSC gap and cardiac and vascular structure and function, with special attention to findings that have been related to prognosis in previous studies.
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An examination of the nature of attentional deficits in patients with Parkinson's disease: evidence from a spatial orienting task.

TL;DR: The results suggest that PD patients may experience a rapid decay of attentional inhibition and do not support the notion that a decrement in processing resources underlies their attentional deficits, but further support the idea that the basal ganglia may play an important role in attentional functions.