M
Masami Ishii
Researcher at University of Tokyo
Publications - 18
Citations - 1430
Masami Ishii is an academic researcher from University of Tokyo. The author has contributed to research in topics: Gene expression & Gene expression profiling. The author has an hindex of 13, co-authored 18 publications receiving 1383 citations. Previous affiliations of Masami Ishii include Kyorin University.
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Journal Article
Differential gene expression profiles of scirrhous gastric cancer cells with high metastatic potential to peritoneum or lymph nodes.
Yoshitaka Hippo,Masakazu Yashiro,Masami Ishii,Hirokazu Taniguchi,Shuichi Tsutsumi,Kosei Hirakawa,Tatsuhiko Kodama,Hiroyuki Aburatani +7 more
TL;DR: This study performs global analysis on differential gene expression of a scirrhous gastric cancer cell line and its derivative sublines with high potential for metastasis to the peritoneal cavity and lymph nodes in a nude mice model and reveals two functional gene clusters with altered expression.
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Targets of transcriptional regulation by two distinct type I receptors for transforming growth factor-beta in human umbilical vein endothelial cells.
Tatsuru Ota,Makiko Fujii,Takashi Sugizaki,Masami Ishii,Keiji Miyazawa,Hiroyuki Aburatani,Kohei Miyazono,Kohei Miyazono +7 more
TL;DR: Microarray analysis of human umbilical vein endothelial cells infected with recombinant adenoviruses carrying a constitutively active form of ALK‐1 or AlK‐5 revealed some new targets of TGF‐β in endothelial Cells, and differences in transcriptional regulation patterns between ALK •1 and ALK•5.
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Direct comparison of GeneChip and SAGE on the quantitative accuracy in transcript profiling analysis.
Masami Ishii,Shin-ichi Hashimoto,Shuichi Tsutsumi,Yoichiro Wada,Kouji Matsushima,Tatsuhiko Kodama,Hiroyuki Aburatani +6 more
TL;DR: GeneChip technology is reasonably reliable for quantitative analysis of expression profiling and would be appropriate as a common platform upon which to build a gene expression database.
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Fish oil feeding alters liver gene expressions to defend against PPARα activation and ROS production
Mayumi Takahashi,Nobuyo Tsuboyama-Kasaoka,Teruyo Nakatani,Masami Ishii,Shuichi Tsutsumi,Hiroyuki Aburatani,Osamu Ezaki +6 more
TL;DR: Analysis of gene expression profile of mice liver indicates that fish oil feeding downregulated the endogenous PPARalpha-activation system and increased antioxidant gene expressions to protect against ROS excess.
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Defect of histone acetyltransferase activity of the nuclear transcriptional coactivator CBP in Rubinstein–Taybi syndrome
Takashi Murata,Riki Kurokawa,Anna Krones,Ken Tatsumi,Masami Ishii,Tomohiko Taki,Mitsuo Masuno,Hirofumi Ohashi,Masayoshi Yanagisawa,Michael G. Rosenfeld,Christopher K. Glass,Yasuhide Hayashi +11 more
TL;DR: The results suggest that the loss of the HAT activity of CBP may cause RTS, as the first example of a defect of H AT activity in a human disease, and raise the possibility that treatment of RTS patients with histone deacetylase inhibitors might have beneficial effects.