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Matthew Rolston

Researcher at University of California, Davis

Publications -  13
Citations -  208

Matthew Rolston is an academic researcher from University of California, Davis. The author has contributed to research in topics: Microbiome & Biology. The author has an hindex of 3, co-authored 7 publications receiving 132 citations. Previous affiliations of Matthew Rolston include University of California.

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Endogenous Enterobacteriaceae underlie variation in susceptibility to Salmonella infection

TL;DR: It is shown that genetically similar laboratory mice obtained from four different commercial vendors exhibited marked phenotypic variation in their susceptibility to Salmonella infection, and endogenous Enterobacteriaceae, a low-abundance taxon, was identified as a keystone species responsible for variation in the susceptibility.
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Analysis of Gut Microbiome and Diet Modification in Patients with Crohn's Disease.

TL;DR: Changes in the composition and complexity of the gut microbiome were identified in response to specialized carbohydrate diet and the SCD was associated with restructuring of the Gut microbial communities.
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Short-Term Western Diet Intake Promotes IL-23‒Mediated Skin and Joint Inflammation Accompanied by Changes to the Gut Microbiota in Mice.

TL;DR: This article showed that exposure to a high-sugar and moderate-fat diet in mice induces appreciable skin inflammation and enhances the susceptibility to imiquimod-induced psoriasiform dermatitis, suggesting that dietary components may render the skin susceptible to psoriatic inflammation.
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Rectal Microbiome Composition Correlates with Humoral Immunity to HIV-1 in Vaccinated Rhesus Macaques.

TL;DR: Assessment of longitudinal changes in vaginal and rectal microbiome profiles after intradermal immunization with a human immunodeficiency virus type 1 (HIV-1) DNA vaccine in adult rhesus macaques shows that the temporal stability of bacterial communities following DNA immunization is site dependent and highlights the importance of host-microbiome interactions in shaping HIV-1 vaccine responses.