Md. Hashim Reza

TL;DR: It is proposed that AT-rich centromeres drive karyotype diversity in the Malassezia species complex through breakage and inactivation, which suggests two distinct mechanisms of chromosome number reduction from an inferred nine-chromosome ancestral state.

TL;DR: Atypical kinetochore dynamics and functional centromeres in M. oryzae are identified and the centromere loci in the top model fungal pathogens that belong to Magnaporthales and cause severe losses in the global production of food crops and turf grasses are identified.

TL;DR: Impairment of development and infectivity of knockdown transformants and altered intracellular cation composition suggest that CorA transporters are essential for Mg2+ homeostasis within the cell, and are crucial to maintaining normal gene expression associated with cell structure, signal transduction and surface hydrophobicity in M. oryzae.

TL;DR: Analysis of synteny conservation across centromeres with newly generated chromosome-level genome assemblies suggests two distinct mechanisms of chromosome number reduction from an inferred 9-chromosome ancestral state: chromosome breakage followed by loss of centromere DNA andCentromere inactivation accompanied by changes in DNA sequence following chromosome-chromosa fusion.

TL;DR: A significant role of glutathione metabolism in the growth and virulence of M. oryzae is suggested and the γ-glutamyl transpeptidase inhibitor severely compromises the vegetative growth of the M.oryzae cells lacking the DUG3 gene.