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Michael Kjaer

Researcher at University of Copenhagen

Publications -  516
Citations -  32647

Michael Kjaer is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Tendon & Skeletal muscle. The author has an hindex of 100, co-authored 494 publications receiving 29502 citations. Previous affiliations of Michael Kjaer include Health Science University & Frederiksberg Hospital.

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Role of the sympathoadrenergic system in adipose tissue metabolism during exercise in humans

TL;DR: It is concluded that circulating catecholamines are important for the exercise‐induced increase in subcutaneous adipose tissue lipolysis while sympathetic nerve activity is not.
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Adrenaline and glycogenolysis in skeletal muscle during exercise: a study in adrenalectomised humans.

TL;DR: It is demonstrated that during exercise muscle glycogenolysis can occur in the absence of adrenaline, and that adrenaline does not enhance muscle glycogensolysis in exercising adrenalectomised subjects and that the activity of HSL in human muscle is increased in exercising man.
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Cardiovascular and neuroendocrine responses to exercise in hypoxia during impaired neural feedback from muscle.

TL;DR: The hypoxia-induced enhancement of systemic adaptation to exercise is not mediated by neural feedback from working muscle in humans.
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Exercise-dependent IGF-I, IGFBPs, and type I collagen changes in human peritendinous connective tissue determined by microdialysis.

TL;DR: The results indicate an increased local production of PICP in human peritendinous tissue in response to prolonged mechanical loading with part of the increase due to trauma from the sampling technique, suggesting that IGFBP-4 may have a key role in the IGF-axis effect on the human collagen synthesis in vivo.
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Acute effects of caffeine ingestion at rest in humans with impaired epinephrine responses

TL;DR: The data demonstrate that caffeine in physiological doses directly stimulates specific tissues, i.e., adipose and peripheral vascular tissue, and these effects are not secondary to increases in Epi after caffeine ingestion.