scispace - formally typeset
M

Ming Zhao

Researcher at Ningbo University

Publications -  18
Citations -  271

Ming Zhao is an academic researcher from Ningbo University. The author has contributed to research in topics: Cancer & Randomized controlled trial. The author has an hindex of 8, co-authored 17 publications receiving 192 citations. Previous affiliations of Ming Zhao include The University of Nottingham Ningbo China.

Papers
More filters
Journal ArticleDOI

Dipeptidyl peptidase-4 inhibitors and cancer risk in patients with type 2 diabetes: a meta-analysis of randomized clinical trials.

TL;DR: The results of this meta-analysis suggest that patients with type 2 diabetes treated with DPP4i do not have a higher risk of developing cancers than patients treated with a placebo or other drugs.
Journal ArticleDOI

HMGA2 promotes adipogenesis by activating C/EBPβ-mediated expression of PPARγ.

TL;DR: It is demonstrated that high mobility group protein AT-hook 2 is required for C/EBPβ-mediated expression of PPARγ, and thus promotes adipogenic differentiation, and indicates that HMGA2 is an essential constituent of the adipogenic transcription factor network, and therefore its function may be affected during the course of obesity.
Journal ArticleDOI

Prevalence of Pre-Diabetes and Its Associated Risk Factors in Rural Areas of Ningbo, China.

TL;DR: The rural areas in Ningbo had a high prevalence of pre-diabetes, and overweight and obesity, hypertension, and elevated triglycerides were the major risk factors.
Journal ArticleDOI

Novel long non-coding RNA GACAT3 promotes gastric cancer cell proliferation through the IL-6/STAT3 signaling pathway

TL;DR: It is demonstrated that as a downstream target of the IL6/STAT3 signaling, lncRNA GACAT3 promotes gastric cancer cell growth suggesting that GACat3 is an inflammatory response gene and may be served as a valuable potential target for the treatment of Gastric cancer.
Journal ArticleDOI

Obesity-associated digestive cancers: A review of mechanisms and interventions:

TL;DR: Evidence that obesity-related gene mutations, insulin and insulin-like growth factor signaling pathways, chronic inflammation, and altered adipokine levels often function interdependently rather than independently in carcinogenesis is presented.