M
Mingsong Wang
Researcher at Shanghai Jiao Tong University
Publications - 27
Citations - 641
Mingsong Wang is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Lung cancer & Metastasis. The author has an hindex of 11, co-authored 26 publications receiving 475 citations.
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Journal ArticleDOI
Overactivated Neddylation Pathway as a Therapeutic Target in Lung Cancer
Lihui Li,Mingsong Wang,Guangyang Yu,Ping Chen,Ping Chen,Hui Li,Dongping Wei,Ji Zhu,Li Xie,Huixun Jia,Jieyi Shi,Chunjie Li,Wantong Yao,Yanchun Wang,Qiang Gao,Lak Shin Jeong,Lak Shin Jeong,Hyuk Woo Lee,Jinha Yu,Fengqing Hu,Ju Mei,Ping Wang,Yiwei Chu,Hui Qi,Meng Yang,Ziming Dong,Yi Sun,Robert M. Hoffman,Lijun Jia +28 more
TL;DR: The overactivated neddylation pathway in lung cancer development and as a promising therapeutic target is highlighted in both lung adenocarcinoma and squamous-cell carcinoma.
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miR-342-3p targets RAP2B to suppress proliferation and invasion of non-small cell lung cancer cells
TL;DR: Preliminary evidence is provided that miR-342-3p acts as a tumor suppressor in NSCLC through repression of RAP2B through interactions with its 3′-UTR region.
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The role of the ubiquitin-proteasome pathway in cancer development and treatment.
TL;DR: The current review focuses on the role of the UPS in cancer development and the development of UPS inhibitors for cancer treatment.
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GLUT5-mediated fructose utilization drives lung cancer growth by stimulating fatty acid synthesis and AMPK/mTORC1 signaling
Wen Lian Chen,Xing Jin,Mingsong Wang,Dan Liu,Qin Luo,Hechuan Tian,Lili Cai,Lifei Meng,Rui Bi,Lei Wang,Xiao Xie,Guanzhen Yu,Lihui Li,Changsheng Dong,Qiliang Cai,Wei Jia,Wenyi Wei,Lijun Jia +17 more
TL;DR: It is demonstrated that, under the condition of coexistence of metabolic fuels in the body, fructose was readily used by LC cells in vivo as a glucose alternative via upregulating GLUT5, a major fructose transporter encoded by solute carrier family 2 member 5 (SLC2A5).
Journal Article
miR-106a promotes growth and metastasis of non-small cell lung cancer by targeting PTEN
TL;DR: This study suggested that miR-106a inhibited the growth and metastasis of NSCLC cells by decreasing PTEN expression, and provided novel insights with potential therapeutic applications for the treatment ofNSCLC.