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Nam Jin Yoo
Researcher at Catholic University of Korea
Publications - 405
Citations - 13537
Nam Jin Yoo is an academic researcher from Catholic University of Korea. The author has contributed to research in topics: Frameshift mutation & Germline mutation. The author has an hindex of 63, co-authored 403 publications receiving 12692 citations. Previous affiliations of Nam Jin Yoo include Catholic University College, Kensington & The Catholic University of America.
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Frameshift mutations of ATBF1, WNT9A, CYLD and PARK2 in gastric and colorectal carcinomas with high microsatellite instability
TL;DR: This study described an additional case of PAMT of the stomach, which shares the clinical, histological, immunophenotypical and ultrastructural features of the previously described cases.
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Identification of large-scale molecular changes of Autotaxin(ENPP2) knock-down by small interfering RNA in breast cancer cells.
Ji Heon Noh,So Yeon Ryu,Jung Woo Eun,Jaehwi Song,Young-Min Ahn,Su Young Kim,Sug Hyung Lee,Won Sang Park,Nam Jin Yoo,Jung Young Lee,Shi Nae Lee,Suk Woo Nam +11 more
TL;DR: The data suggest that the molecular signature identified by the ENPP2-silencing methods may represent potential candidates that can explain the complicated characteristics of ATX and may serve as biomarkers, for the development of molecular-targeting therapy, in human breast cancer.
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Somatic mutation of proapoptotic caspase-2 gene is rare in acute leukemias and common solid cancers.
TL;DR: Cancer tissues from acute leukemias, breast cancers, lung cancers, and liver cancers for the detection of caspase-2 somatic mutations are analyzed and roles of apoptosis deregulation in cancer development led us to analyze caspases' somatic mutation in this study.
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Histone Demethylase Gene PHF2 Is Mutated in Gastric and Colorectal Cancers.
TL;DR: The data reveal that TSG gene PHF2 harbors mutational ITH as well as the frameshift mutations in CRC and GC with MSI-H, which is suggested to play a role in tumorigenesis through its TSG inactivation in CRCand GC.
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Frameshift Mutations of HSPA4 and MED13 in Gastric and Colorectal Cancers
TL;DR: The data show that unconventional HSPA4 and MED13 genes harbored frameshift mutations that might possibly inactivate their functions and could be a feature of GC and CRC with MSI-H.