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Nancy K. Mueller

Researcher at University of Cincinnati

Publications -  10
Citations -  3501

Nancy K. Mueller is an academic researcher from University of Cincinnati. The author has contributed to research in topics: Amygdala & Hypothalamus. The author has an hindex of 8, co-authored 10 publications receiving 3252 citations. Previous affiliations of Nancy K. Mueller include University of Cincinnati Academic Health Center.

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Central mechanisms of stress integration: hierarchical circuitry controlling hypothalamo-pituitary-adrenocortical responsiveness.

TL;DR: The principle extrinsic and intrinsic mechanisms responsible for regulating stress-responsive CRH neurons of the hypothalamic paraventricular nucleus, which summate excitatory and inhibitory inputs into a net secretory signal at the pituitary gland, are reviewed.
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Limbic system mechanisms of stress regulation: hypothalamo-pituitary-adrenocortical axis.

TL;DR: The influence of the limbic system on the HPA axis is likely the end result of the overall patterning of responses to given stimuli and glucocorticoids, with the magnitude of the secretory response determined with respect to the relative contributions of the various structures.
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Role of GABA and Glutamate Circuitry in Hypothalamo‐Pituitary‐Adrenocortical Stress Integration

TL;DR: It seems the psychogenic responses to stress are gated by discrete sets of GABAergic neurons in the basal forebrain and hypothalamus, which may play a major role in HPA dysfunction seen in affective disease states and aging.
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Role of the ventral subiculum in stress integration.

TL;DR: The present state of knowledge indicates that the role of the subiculum in stress integration is complex, and likely involves interactions of stress-relevant subicular output with limbic-hypothalamic stress-integrative circuits.
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Daily limited access to sweetened drink attenuates hypothalamic-pituitary-adrenocortical axis stress responses

TL;DR: The hypothesis that the intake of palatable substances represents an endogenous mechanism to dampen physiological stress responses is supported, as limited consumption of sweetened drink attenuates hypothalamic-pituitary-adrenocortical axis stress responses.