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Nancy R. Rice
Researcher at National Institutes of Health
Publications - 49
Citations - 4695
Nancy R. Rice is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Virus & Gene. The author has an hindex of 34, co-authored 49 publications receiving 4616 citations. Previous affiliations of Nancy R. Rice include University of Michigan.
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Role of NF-κB in p53-mediated programmed cell death
TL;DR: It is concluded that inhibition of NF-κB in tumours that retain wild-type p53 may diminish, rather than augment, a therapeutic response.
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Induction of In Vitro Reprogramming by Toll-Like Receptor (TLR)2 and TLR4 Agonists in Murine Macrophages: Effects of TLR “Homotolerance” Versus “Heterotolerance” on NF-κB Signaling Pathway Components
Marina A. Dobrovolskaia,Andrei E. Medvedev,Karen E. Thomas,Natalia Cuesta,Vladimir Y. Toshchakov,Tianbo Ren,Michael J. Cody,Suzanne M. Michalek,Nancy R. Rice,Stefanie N. Vogel +9 more
TL;DR: It is demonstrated that induction of homotolerance affects a broader spectrum of signaling components than in heterotolerance, with selective modulation of specific elements within the NF-κB signaling pathway.
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Myristylation site in Pr65gag is essential for virus particle formation by moloney murine leukemia virus
TL;DR: The results are consistent with the theory that myristate is required for the association of the gag protein with the plasma membrane and that this association is necessary for virus assembly.
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E2F-1 potentiates cell death by blocking antiapoptotic signaling pathways.
TL;DR: It is shown that E2F-1 can induce apoptosis by a death receptor-dependent mechanism, by downregulating TRAF2 protein levels and inhibiting activation of antiapoptotic signals including NF-kappa B.
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Equine infectious anemia virus gag and pol genes: relatedness to visna and AIDS virus.
TL;DR: The nucleotide sequence of the gag and pol genes of an equine infectious anemia virus (EIAV) proviral DNA clone was determined and these viruses constitute a family clearly distinct from that of the type C viruses or the BLV-HTLV-I and -II group.