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Nathalie Charnaux

Researcher at University of Paris

Publications -  70
Citations -  2797

Nathalie Charnaux is an academic researcher from University of Paris. The author has contributed to research in topics: Chemokine & Syndecan 1. The author has an hindex of 31, co-authored 67 publications receiving 2564 citations. Previous affiliations of Nathalie Charnaux include Sorbonne & French Institute of Health and Medical Research.

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The shedding of syndecan-4 and syndecan-1 from HeLa cells and human primary macrophages is accelerated by SDF-1/CXCL12 and mediated by the Matrix Metalloproteinase-9

TL;DR: It is demonstrated that SDF-1 strongly accelerates the shedding of syndecan-4 ectodomains and to a lesser extent that of syndECan-1 from HeLa cells, which strongly indicates the role of MMP-9 in these events occurring in both a tumoral cell line and in human primary macrophages.
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Determinants of Vitamin D Status in Caucasian Adults: Influence of Sun Exposure, Dietary Intake, Sociodemographic, Lifestyle, Anthropometric, and Genetic Factors

TL;DR: Several modifiable factors were identified, such as daily-life moderate sun Exposure, physical activity, and normal-weight maintenance, which should be targeted by public health policies in order to improve vitamin D status in the general population, while avoiding active/intensive sun exposure, in line with recommendations for skin cancer prevention.
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Stromal cell-derived factor-1/chemokine (C-X-C motif) ligand 12 stimulates human hepatoma cell growth, migration, and invasion.

TL;DR: The fact that CXCR4 is expressed in hepatocellular carcinoma cells from liver biopsies indicates that the in vitro results reported here could be extended to in vivo conditions and strongly indicates that SDC-4 may be an auxiliary receptor for SDF-1.
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Follicle-Stimulating Hormone Receptors in Oocytes?

TL;DR: Observations concur to demonstrate the presence of functional FSH receptors in oocytes and raise the possibility of direct control of oocyte development by FSH.
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Liver iron, HFE gene mutations, and hepatocellular carcinoma occurrence in patients with cirrhosis.

TL;DR: Liver iron overload and C282Y mutation are associated with a higher risk of HCC in patients with alcoholic but not HCV-related cirrhosis and remain independent risk factors for HCC.